Western celery mosaic

Severin, Henry H. P.; Freitag, Julius H.

doi:10.3733/hilg.v11n09p493

Cited by 52 publications

(35 citation statements)

References 9 publications

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“…DISCUSSION Certain symptoms of the Australian carrot disease resemble those described for the American diseases caused by the Californian Aster Yellows (Severin 1932) and Western Celery Mosaic (Severin and Freitag 1938) viruses, but there seems to be little doubt that the local virus is distinct from either of these viruses.…”

Section: A New Virus Disease Of Carrots 329mentioning

confidence: 95%

“…Severin and Freitag (1938) also recorded transmission of Western Celery Mosaic virus with this aphid, which bred on celery.…”

Section: (E) Previous Records Of Virus Transmission By Cavariella Aegmentioning

confidence: 99%

“…At that time virus diseases of carrots had only been recorded in the United States, where both the Aster Yellows virus (Zundel 1929;Severin 1930) and the Western Celery Mosaic virus (Severin and Freitag 1938) caused economically imp()rtant diseases. Neither of these viruses had been recorded in Australia.…”

Section: Identification Of the Diseasementioning

confidence: 99%

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A New Virus Disease of Carrots: Its Transmission, Host Range, and Control

Stubbs¹

1948

Aust. Jnl. Of Bio. Sci.

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Section: A New Virus Disease Of Carrots 329mentioning

confidence: 95%

“…Severin and Freitag (1938) also recorded transmission of Western Celery Mosaic virus with this aphid, which bred on celery.…”

Section: (E) Previous Records Of Virus Transmission By Cavariella Aegmentioning

confidence: 99%

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A New Virus Disease of Carrots: Its Transmission, Host Range, and Control

Stubbs¹

1948

Aust. Jnl. Of Bio. Sci.

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“…It transmitted beet mosaic once in some hundreds of experiments (Severin and Drake 1948) and transmitted western celery mosaic in only 2· 2 per cent. of tests (Severin and Freitag 1938); these performances indicate an unusual inability to act as a vector and suggest that this species would repay careful study. A comparable, though less marked result was reported by Watson and Roberts (1939) for M. euphorbiae (= M. gei).…”

Section: ( D) What Explanations Are Available For the Observed Specifmentioning

confidence: 99%

On the Mechanism of Transmission of Non-Persistent Phytopathogenic Viruses by Aphids

Day¹,

Irzykiewicz²

1954

Aust. Jnl. Of Bio. Sci.

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The following is a working hypothesis, based on published data, to eluddate the mechanism of transmission of n9n-persistent viruses by aphids: The stylets of the vector become contaminated with virus during probing in infected tissues. Only this virus is of significance in disease transmission, and ingested virus plays no part in the process. Some of the virus on the stylets is inactivated by salivary fluids. Viruses differ in their susceptibility to this inhibition and different species of aphids vary in the production of the inhibitor. Fasting decreases the activity of the salivary inhibitors.

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“…Electron microscopy of leaf samples of cow parsley often revealed rather rigid elongate virus particles in low concentrations. The virus was Murant and Goold, 1968;Elnagar and Murant, 1976this paper Murant, 1974bWaterhouse and Murant, 1982Bos, 1985 Yamashita et al, 1976; this paper Bos et al, 1978 Shepard andGrogan, 1971;Bos et al, 1989 Severin andFreitag, 1938;Hollings, 1964;Pemberton and Frost, 1986; this paper Wolf, 1972b;this paper Bem andMurant, 1979b Bern andMurant, 1979a; see also parsnip leafcurl virus (this paper) Bern and Murant, 1979a;Murant et al, 1984; see also carrot yellow leaf virus (this paper) Bos et al, 1979 see hogweed virus 4; this paper Murant, 1972; Watson et al, 1964;this paper Murant, 1974a;Van Dijk and Bos, 1985Murant, 1974allarvirus Fulton, 1985 …”

Section: Resultsmentioning

confidence: 99%