Wallenberg's Syndrome With Proximal Quadriparesis

Chen, Chau-Nee; Khor, Gim-Thean; Chen, Chun‐Hung; Huang, Poyin

doi:10.1097/nrl.0b013e3181ebe5b2

The Neurologist

2011

DOI: 10.1097/nrl.0b013e3181ebe5b2

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Wallenberg's Syndrome With Proximal Quadriparesis

Chau-Nee Chen¹,

Gim-Thean Khor²,

Chun‐Hung Chen³

et al.

Abstract: the proximal quadriparesis can be explained by involvement of the right medial medullary reticular formation. Our observation supports the hypothesis that the proximal part of the limb is bilaterally innervated by axons of the corticoreticulospinal tracts in human beings. The rapid recovery of proximal quadriparesis also supports the concept that a functional pyramidal tract seems crucial for recovery after stroke.

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Cited by 3 publications

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Targeted Loss of GHR Signaling in Mouse Skeletal Muscle Protects Against High-Fat Diet–Induced Metabolic Deterioration

Vijayakumar

Sun

et al. 2011

Diabetes

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Growth hormone (GH) exerts diverse tissue-specific metabolic effects that are not revealed by global alteration of GH action. To study the direct metabolic effects of GH in the muscle, we specifically inactivated the growth hormone receptor (ghr) gene in postnatal mouse skeletal muscle using the Cre/loxP system (mGHRKO model). The metabolic state of the mGHRKO mice was characterized under lean and obese states. High-fat diet feeding in the mGHRKO mice was associated with reduced adiposity, improved insulin sensitivity, lower systemic inflammation, decreased muscle and hepatic triglyceride content, and greater energy expenditure compared with control mice. The obese mGHRKO mice also had an increased respiratory exchange ratio, suggesting increased carbohydrate utilization. GH-regulated suppressor of cytokine signaling-2 (socs2) expression was decreased in obese mGHRKO mice. Interestingly, muscles of both lean and obese mGHRKO mice demonstrated a higher interleukin-15 and lower myostatin expression relative to controls, indicating a possible mechanism whereby GHR signaling in muscle could affect liver and adipose tissue function. Thus, our study implicates skeletal muscle GHR signaling in mediating insulin resistance in obesity and, more importantly, reveals a novel role of muscle GHR signaling in facilitating cross-talk between muscle and other metabolic tissues.

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Targeted Loss of GHR Signaling in Mouse Skeletal Muscle Protects Against High-Fat Diet–Induced Metabolic Deterioration

Vijayakumar

Sun

et al. 2011

Diabetes

View full text Add to dashboard Cite

show abstract

The distribution of the cortical origin of the corticoreticular pathway in the human brain: A diffusion tensor imaging study

Jang¹,

Seo²

2014

Somatosensory & Motor Research

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We investigated the distribution of the cortical origin of the corticoreticular pathway (CRP) in the human brain. Forty normal subjects were recruited and CRPs from four cortical areas were reconstructed. The first cortical origin area of the CRP was the premotor cortex and the next was the primary motor cortex. Although the CRP fibers also originated from the primary somatosensory cortex and prefrontal cortex, they occupied the smallest portion among four regions of interest.

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An unusual case of posterior circulation stroke

Tharaknath¹,

Chowdary²,

Kumar³

et al. 2017

J NTR Univ Health Sci

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Wallenberg's Syndrome With Proximal Quadriparesis

Cited by 3 publications

References 0 publications

Targeted Loss of GHR Signaling in Mouse Skeletal Muscle Protects Against High-Fat Diet–Induced Metabolic Deterioration

Targeted Loss of GHR Signaling in Mouse Skeletal Muscle Protects Against High-Fat Diet–Induced Metabolic Deterioration

The distribution of the cortical origin of the corticoreticular pathway in the human brain: A diffusion tensor imaging study

An unusual case of posterior circulation stroke

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