Vertigo in Cerebrovascular Disease

Cm, Fisher

doi:10.1001/archotol.1967.00760040531010

Cited by 138 publications

(67 citation statements)

References 8 publications

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“…Transient vertebrobasilar ischaemia is a difficult diagnosis and unlikely to be correct in a patient who has attacks consisting only of vertigo. 46 Nonetheless, it could be suspected in a patient in whom only some of the vertigo attacks are accompanied by other symptoms of brainstem dysfunction such as diplopia. [47][48][49] In the absence of any simultaneous brainstem symptoms, a short history, of days rather than months, of frequent brief vertigo attacks lasting minutes rather than hours several times a day, should raise the suspicion that the attacks are posterior circulation transient ischaemic attacks (TIAs).…”

Section: Diagnoses That Are Likely To Be Wrong In a Patient With Vertigomentioning

confidence: 99%

Diagnosis and management of vertigo

Halmagyi

2005

Clinical Medicine

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-Vertigo is an illusion of rotation due to a disorder of the vestibular system, almost always peripheral. In the history it must be distinguished from pre-syncope, seizures and panic attacks. A single attack of acute, isolated spontaneous vertigo lasting a day or more is due either to vestibular neuritis or cerebellar infarction; distinguishing between the two requires mastery of the head impulse test. Recurrent vertigo is mostly due to benign paroxysmal positioning vertigo (BPPV), Menière's disease or migraine. With a good history, a positional test, an audiogram and a caloric test, it is usually possible to distinguish between these. BPPV is the single most common cause of recurrent vertigo and can usually be cured immediately with a particle repositioning manoeuvre. Posterior circulation ischaemia very rarely causes isolated vertigo attacks and when it does the attacks are brief and frequent and the history is short. KEY WORDS What vertigo is and is notVertigo is an illusion of rotation caused by asymmetry of neural activity between the left and right vestibular nuclei. This can be induced in anyone• by simply spinning them at constant speed for about 20 seconds and then suddenly stopping them• by heating or cooling one labyrinth, as happens in the caloric test• by otoconial particles (calcium-containing debris: literally 'earstones') moving along semicircular canal duct, as happens in benign paroxysmal positional vertigo (BPPV). It can also be produced by sudden unilateral destruction of a normal vestibular end-organ, nerve or nucleus or vestibulo-cerebellum, a structure that normally inhibits the ipsilateral vestibular nucleus. (Bilateral simultaneous vestibular destruction does not produce vertigo -in fact, a patient with bilateral vestibular loss will never again experience vertigo from unilateral or asymmetrical vestibular stimulation.)Vertigo is always temporary. Even after the vestibular nerve on one side has been surgically severed, the resulting vertigo (and nystagmus) will abate within a few days. This is not because the vestibular nerve has re-anastomosed but because profound neurochemical changes occuring in the brainstem during the process of vestibular compensation restore symmetrical vestibular nucleus activity. Vertigo is always made worse by head movement; thus patients who are 'dizzy all the time' and are happy to move around whilst dizzy do not have vertigo.Patients with aural vertigo should not lose consciousness but some patients cannot give confident and convincing answers to such simple questions as 'Did it feel as if you were losing balance or as if you were losing consciousness? Did you feel as if you were going to pass out or fall over?' Patients with vertigo might lose consciousness if they have been vomiting a lot, or if they incurred a head injury when they fell.Witness descriptions are not much help in identifying vertigo, but can be helpful in identifying other paroxysmal disorders such as seizure and syncope. Whereas panic attacks, especially with hyperventilation, co...

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Section: Diagnoses That Are Likely To Be Wrong In a Patient With Vertigomentioning

confidence: 99%

Diagnosis and management of vertigo

Halmagyi

2005

Clinical Medicine

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“…Semptomların geçici olması nedeniyle VBY tanısını koymak oldukça zordur. Vertigo nadiren VBY'nin tek bulgusu olur 50 . Vertebrobaziler yetmezlik tek başına vertigoya sebep olabileceği gibi yutma güçlüğü, konuşma güçlüğü, çift taraflı görme bozuklukları ve çift görmeyle birlikte de olabilir.…”

Section: Vertebrobaziler Yetmezlikunclassified

Acil Serviste Vertigoya Yaklaşım

Koçer¹,

Avcı²,

Karakısa³

et al. 2017

Arşiv Kaynak Tarama Dergisi

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Vertigo is one of the frequent causes of admissions to emergency services. Vertigo and dizziness should be distinguished on admission. It should be well known that dizziness might based on more underlying problems with vital abnormalities compared to vertigo. Medication reduces the symptoms and mainstay of treatment is vestibular tests. The aims of this review are to distinguish central and peripheral vertigo, evaluate approaches to discharge patients form the emergency room with simple maneuvers and describe general approach to the patients that require further evaluation and treatment.

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“…Fisher [50] w grupie 112 chorych z niewydolnością krążenia podstawnego, "ostry" niedosłuch wykazał u 10 z nich (8,9%). Wyższy odsetek nagłych głuchot niż w prezentowanych powyżej doniesieniach podali Grad i wsp.…”

Section: Historia Badań Nad Objawami Otologicznymi W Udarze Niedokunclassified

“…[33] opis całkowitej głuchoty u kobiety z obustronnym udarem torebki wewnętrznej (1 badany) 1943 Adams R. D. [2] pierwszy opis zespołu objawów związanych z zamknięciem tętnicy móżdżko-wej przedniej dolnej, w którym występowały: zawroty głowy, niedosłuch, niedowład nerwu twarzowego, ataksja, oczopląs i niedoczulica (1 badany) 1962 Williams D. i wsp. [229] niedosłuch u 1,5% chorych z udarem w zakresie unaczynienia krążenia podstawnego (65 badanych) 1967 Fisher C. M. [50] "ostry niedosłuch" u 8,9% chorych z udarem i niewydolnością krążenia podstawnego (112 badanych) 1970 Ruszel J. [183] opis przypadku nagłej głuchoty w przebiegu udaru pniowego, którego przyczyną był potwierdzony badaniem sekcyjnym zakrzep t ę tnicy podstawnej (1 badany) 1989 Grad A. i wsp.…”

Section: Historia Badań Nad Objawami Otologicznymi W Udarze Niedokunclassified

Stan słuchu u chorych we wczesnym okresie udaru niedokrwiennego mózgu

Przewoźny¹

2007

Otolaryngologia Polska

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Vertigo in Cerebrovascular Disease

Cited by 138 publications

References 8 publications

Diagnosis and management of vertigo

Diagnosis and management of vertigo

Acil Serviste Vertigoya Yaklaşım

Stan słuchu u chorych we wczesnym okresie udaru niedokrwiennego mózgu

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