2017
DOI: 10.1155/2017/9680891
|View full text |Cite
|
Sign up to set email alerts
|

Ventricular Septal Dissection Complicating Inferior Wall Myocardial Infarction

Abstract: Postmyocardial infarction ventricular septal defect is an increasingly rare mechanical complication of acute myocardial infarction. We present a case of acute myocardial infarction from right coronary artery occlusion that developed hypotension and systolic murmur 12 hours after successful percutaneous coronary intervention. Although preoperative imaging suggested a large ventricular septal defect and a pseudoaneurysm, intraoperative findings concluded a serpiginous dissection of the ventricular septum. The im… Show more

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
4
1

Citation Types

0
11
0

Year Published

2021
2021
2024
2024

Publication Types

Select...
5

Relationship

0
5

Authors

Journals

citations
Cited by 5 publications
(12 citation statements)
references
References 4 publications
0
11
0
Order By: Relevance
“…11 To our knowledge, this is one of the very few case reports in the literature of a patient suffering an acute coronary syndrome with normal coronary arteries and subsequent VSR 24 h after catheterization and 30 h after hospital admission. [11][12][13][14][15] Possible explanations for the VSR after the acute coronary event may be either spasm or plaque erosion (more common in NSTEMI, younger age and absence of the 'traditional' cardiovascular risk factors) or paroxysmal atrial fibrillation, that led to transient epicardial thrombosis and/or occlusion or an ostial and isolated occlusion of a large septal perforator that could not be visualized during the coronary angiography procedure.…”
Section: Discussionmentioning
confidence: 99%
“…11 To our knowledge, this is one of the very few case reports in the literature of a patient suffering an acute coronary syndrome with normal coronary arteries and subsequent VSR 24 h after catheterization and 30 h after hospital admission. [11][12][13][14][15] Possible explanations for the VSR after the acute coronary event may be either spasm or plaque erosion (more common in NSTEMI, younger age and absence of the 'traditional' cardiovascular risk factors) or paroxysmal atrial fibrillation, that led to transient epicardial thrombosis and/or occlusion or an ostial and isolated occlusion of a large septal perforator that could not be visualized during the coronary angiography procedure.…”
Section: Discussionmentioning
confidence: 99%
“…However, there have been few reports of post-myocardial infarction ventricular septal dissection forming a septal aneurysm without a shunt [ 5 , 6 ]. Furthermore, to the best of our knowledge, only one case report described a case of a patient who underwent surgical repair of ventricular dissection unaccompanied by a shunt [ 7 ]. This type of pathology is different from other types of post-myocardial infarction complications, such as left ventricular aneurysm or pseudoaneurysms, which we encountered frequently.…”
Section: Discussionmentioning
confidence: 99%
“…In rare cases, a shunt is created by peeling and perforation of the interventricular septum, termed ventricular dissection [ 1 4 ]. However, there have been few reports of post-myocardial infarction ventricular septal dissection unaccompanied by a shunt [ 5 7 ]. We herein report the case of a patient with post-myocardial infarction ventricular septal dissection forming a septal aneurysm that caused cardiogenic shock.…”
Section: Introductionmentioning
confidence: 99%
“…However, a few different hypotheses have been proposed. One of the common hypotheses is that after the MI, the blood supply is compromised, especially in the septal perforator arteries [ 3 ]. Septal perforator arteries are vessels that arise from the right coronary and the LAD artery.…”
Section: Discussionmentioning
confidence: 99%
“…Septal perforator arteries are vessels that arise from the right coronary and the LAD artery. The disruption in the blood supply causes opposing forces from the RCA and LAD on the ventricular septum, thereby resulting in the dissection [ 3 ]. The helical myocardial muscular band also plays a significant role in IVSD [ 2 ].…”
Section: Discussionmentioning
confidence: 99%