Ventricular extrasystoles induced by epinephrine, nicotine, ethanol, and vasopressin in dogs with myocardial lesions

Balázs, T.; Ohtake, Shinzaburo; Cummings, John R.; Noble, John F.

doi:10.1016/0041-008x(69)90147-1

Cited by 11 publications

(2 citation statements)

References 14 publications

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“…Comparable cardiovascular lesions in dogs were first described by Luginbühl and Detweiler in 1965. In 1969, Balazs et al described papillary muscle necrosis after administration of large doses of isoproterenol, a well-known compound used to lower blood pressure. The term myocardial papillary muscle necrosis in dogs was introduced by Balazs and Payne in 1971, after administration of hypotensive compounds and has since been cited in some medical textbooks as the "Balazs phenomenon."…”

Section: Cardiac Papillary Muscle Lesionsmentioning

confidence: 98%

Proceedings of the 2012 National Toxicology Program Satellite Symposium

et al. 2012

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The 2012 annual National Toxicology Program (NTP) Satellite Symposium, entitled “Pathology Potpourri,” was held in Boston in advance of the Society of Toxicologic Pathology’s 31st annual meeting. The goal of the NTP Symposium is to present current diagnostic pathology or nomenclature issues to the toxicologic pathology community. This article presents summaries of the speakers’ presentations, including diagnostic or nomenclature issues that were presented, along with select images that were used for audience voting or discussion. Some lesions and topics covered during the symposium include eosinophilic crystalline pneumonia in a transgenic mouse model; differentiating adrenal cortical cystic degeneration from adenoma; atypical eosinophilic foci of altered hepatocytes; differentiating cardiac schwannoma from cardiomyopathy; diagnosis of cardiac papillary muscle lesions; intrahepatocytic erythrocytes and venous subendothelial hepatocytes; lesions in Rathke’s cleft and pars distalis; pernicious anemia and megaloblastic disorders; embryonic neuroepithelial dysplasia, holoprosencephaly and exencephaly; and INHAND nomenclature for select cardiovascular lesions.

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Section: Cardiac Papillary Muscle Lesionsmentioning

confidence: 98%

Proceedings of the 2012 National Toxicology Program Satellite Symposium

et al. 2012

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“…In animals fed normal diets, ethanol has produced conduction delays [6] and paroxysmal extrasystoles after induced myocardial necrosis [7]. Studies with in vitro preparations of the heart have shown opposite concentration-related actions on force and rate [8,9].…”

Section: Introductionmentioning

confidence: 99%

Ethanol as an arrhythmogen and an antiarrhythmic agent with reoxygenation arrhythmias of cultured heart cells

Wenzel

Fuh

1981

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Cultures of neonatal rat heart myoblasts exposed to constant ethanol concentrations of 26, 64, 160 or 400 mg% for 3 days were more arrhythmle during this time than control cultures. Although cell growth was not measurably affected, the evidence suggested that arrhythmlas may have developed from impaired eell-to-ceU contact. Exposure of these treated cells to an arrhythmogenle procedure involving hypoxia and reoxygenatlon, demonstrated a modest antiarrhythmie action of ethanol.

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How to Induce Arrhythmias with Isoprenaline

Pripon

2019

Arrhythmia Induction in the EP Lab

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Ventricular extrasystoles induced by epinephrine, nicotine, ethanol, and vasopressin in dogs with myocardial lesions

Cited by 11 publications

References 14 publications

Proceedings of the 2012 National Toxicology Program Satellite Symposium

Proceedings of the 2012 National Toxicology Program Satellite Symposium

Ethanol as an arrhythmogen and an antiarrhythmic agent with reoxygenation arrhythmias of cultured heart cells

How to Induce Arrhythmias with Isoprenaline

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