Vascular interventions for impotence: Lessons learned

Abstract

With prospective screening criteria, 6% to 7% of impotent men became candidates for vascular intervention. Including those functioning with ICI or vacuum constriction devices, about 70% of these men were functional after operation. Men undergoing aortoiliac reconstruction has a significantly higher rate (58%) of spontaneous function as compared with those undergoing microvascular procedures.

Vascular surgery for impotence: a review

“…Using these principles even older patients will exhibit improved erectile function as was mentioned previously. 6,7,41,43,44 Femoro-femoral bypass in certain cases causes similar results. 45 Experience with direct hypogastric artery reconstruction, a relatively rare operation, has been described 46 and has yielded good results.…”

“…6,59 The appearance of glans hyperemia may be delayed as long as 42 months postoperatively. 60 This complication demands immediate attention, as ulceration, tissue loss and urinary retention occur.…”

“…In vascular surgery for impotence, graft closure is often signaled by sudden loss of function. 6 In some instances erectile function does return just as limbs are salvaged after a period of perfusion followed by graft failure. This is also true of deep dorsal vein arterialization.…”

Sexual function after aortic surgery

“…De Palma et al . investigated 1094 male subjects whose chief complaint was impotence, with penile plethysmography and penile brachial indices 13 . Sixty‐seven who were considered to have significant arterial disease were submitted to operation.…”

Operative Therapie der erektilen Dysfunktion

“…Die Folge des vermehrten venösen Abstroms ist eine rasche Detumeszenz, da die Erektion nicht aufrechterhalten werden kann.Aufgrund der Aufsehen erregenden Entwicklung der Pharmakotherapie, anfangs mit den intrakavernösen Injektionen,gefolgt von den potenten oralen Phosphodiesterasehemmern,hat die penile Venenchirurgie heutzutage ihre Bedeutung verloren.Darüber hinaus wissen wir heute, dass der Pathomechanismus im Schwellkörpergewebe bzw. an der Tunica albuginea zu suchen ist [1,4,6,8,14,19,21,22,25].…”