2005
DOI: 10.1016/j.ccl.2005.04.004
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Vascular Access in Hemodialysis: Issues, Management, and Emerging Concepts

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Cited by 88 publications
(102 citation statements)
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References 153 publications
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“…Tingkat infeksi pada fistula arteriovenosa (AV) adalah yang terendah (OR 2,2), di lebih tinggi pada penggunaan kateter double lumen (OR 13,6), dan paling tinggi pada kateter sementara (OR32,6). 29 Pada penelitian ini sebagian besar (89,9%) pasien menjalani HD dengan akses femoral, sementara sisanya menggunakan kateter double lumen dan cimino (8,5% dan 1,6%). Hal serupa juga didapatkan oleh Metcalfe dkk.…”
Section: Penelitian Tahun Nilai Batas Hemoglobin (G/dl)unclassified
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“…Tingkat infeksi pada fistula arteriovenosa (AV) adalah yang terendah (OR 2,2), di lebih tinggi pada penggunaan kateter double lumen (OR 13,6), dan paling tinggi pada kateter sementara (OR32,6). 29 Pada penelitian ini sebagian besar (89,9%) pasien menjalani HD dengan akses femoral, sementara sisanya menggunakan kateter double lumen dan cimino (8,5% dan 1,6%). Hal serupa juga didapatkan oleh Metcalfe dkk.…”
Section: Penelitian Tahun Nilai Batas Hemoglobin (G/dl)unclassified
“…Di sisi lain, kesiapan akses vaskular saat HD pertama merupakan faktor yang bermakna pada mortalitas. 29 Hal ini dapat menjadi alasan mengapa inisiasi HD menjadi tidak bermakna. Dengan demikian perencanaan HD yang baik, terutama persiapan akses vaskular, tetap menjadi faktor penting terhadap mortalitas.…”
Section: Penelitian Tahun Nilai Batas Hemoglobin (G/dl)unclassified
“…Arteriovenous (AV) fistulas have a much higher suitability failure (primary failure) than do AV grafts. However, once suitability for dialysis has been achieved, fistulas have a better cumulative survival as compared with AV grafts (4,5). The main cause of fistula and graft failure is aggressive neointimal hyperplasia that results in stenosis and subsequent thrombosis, most commonly in the perianastomotic region for fistulas or at the venous anastomosis for grafts (6,7).…”
mentioning
confidence: 99%
“…At a histological level early AVF failure is also characterized by aggressive neointimal hyperplasia in both animal and human models, seen as early as 1 month in animals 63,71 and 3 months in humans 64,66 . The underlying factors (upstream events) which may contribute to early AVF failure, include 4,[72][73][74][75][76][77][78][79][80][81] : (1) small diameter sizes in the vein and artery, (2) surgical injury at the time AV fistula placement, (3) previous venipunctures, (4) development of accessory veins after surgery, (5) hemodynamic shear stress at the AV anastomosis, (6) a genetic predisposition to vascular constriction and neointimal hyperplasia, and (7) pre-existing venous neointimal hyperplasia. The subsequent sections will focus on the downstream events and three main mechanisms responsible for neointimal hyperplasia such as oxidative stress, inflammation, endothelial dysfunction, and alternative origins of neointimal-derived cells.…”
Section: Pathophysiologic Mechanisms Of Neointimal Hyperplasia Formatmentioning
confidence: 99%