Use of Commercial Record Linkage Software and Vital Statistics to Identify Patient Deaths

Newman, Thomas B.; Brown, Adrienne S.

doi:10.1136/jamia.1997.0040233

Cited by 189 publications

(105 citation statements)

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“…This was close to the odds ratio for ANLL reported by Altmann et al 12 (odds ratio of 11.1). Narod et al 8 also reported a higher-than-expected rate of congenital abnormalities in children with cancer.…”

Section: Discussionsupporting

confidence: 73%

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Congenital abnormalities and childhood cancer

Agha

Williams

Marrett

et al. 2005

Cancer

129

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BACKGROUNDThe examination of specific characteristics of neoplasms diagnosed in children have suggested that a significant proportion can be attributed to a genetic mutation or genetic predisposition. Although the study of a genetic predisposition to cancer in children remains in the early stages, congenital abnormalities could provide essential information for mapping predisposing lesions in children with cancer.METHODSIn the current study, 2 large cohorts of children with and without congenital abnormalities were followed for the occurrence of cancer and death up to 18 years. Through this study, the risk of developing cancer by age at diagnosis, effects of birth characteristics on cancer risk, and possible associations between specific anomalies and tumor types were examined.RESULTSBased on the follow‐up of 90,400 children, the risk of developing cancer during the first year of life was found to be nearly 6 times higher in children with anomalies (rate ratio [RR] of 5.8; 95% confidence interval [95% CI], 3.7–9.1). Children with birth defects were found to be at a higher risk for developing leukemia (RR of 2.7; 95% CI, 2.1–3.6), tumors of the central nervous system (RR of 2.5; 95% CI, 1.8–3.4), sympathetic nervous system tumors (RR of 2.2; 95% CI, 1.4–3.4), and soft tissue sarcomas (RR of 1.9; 95% CI, 1.0–3.5). Among children with birth defects, children with Down syndrome, nervous system anomalies, and anomalies of the urinary system had the highest incidence rates of cancer. In the presence of birth defects, other factors such as birth weight, gestational age, age of the mother, and birth order were not found to be associated significantly with the risk of cancer.CONCLUSIONSThe significant relative risks found in the current study provided evidence of links between the presence of abnormalities and the development of cancer. Some “cancer‐prone” abnormalities were identified in the current study. Such anomalies may be markers of other exposures or processes that increase the risk of developing cancer. Cancer 2005. © 2005 American Cancer Society.

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Section: Discussionsupporting

confidence: 73%

“…11 It has been reported previously that hospital data can be accurately linked with state and national vital statistics using this record linkage software. 12 …”

Section: Linkagesmentioning

confidence: 99%

Congenital abnormalities and childhood cancer

Agha

Williams

Marrett

et al. 2005

Cancer

129

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“…Death was identified from health plan administrative data (including proxy reporting), California death certificate data, and Social Security Administrative vital status files. 35,36 The primary outcome of interest was occurrence of incident renal cancer during the study period identified from the Kaiser Permanente Cancer Registry. Renal cancer histology codes used in our dataset were in compliance with the World Health Organization International Classification of Diseases for Oncology international standard for primary site and histology (December 2009; www.seer.cancer.gov/ icd-o-3/index.html).…”

Section: Follow-up and Outcomesmentioning

confidence: 99%

CKD and the Risk of Incident Cancer

Lowrance

Ordoñez

Udaltsova

et al. 2014

Journal of the American Society of Nephrology

216

183

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Previous studies report a higher risk of cancer in patients with ESRD, but the impact of less severe CKD on risk of cancer is uncertain. Our objective was to evaluate the association between level of kidney function and subsequent cancer risk. We performed a retrospective cohort study of 1,190,538 adults who were receiving care within a health care delivery system, had a measurement of kidney function obtained between 2000 and 2008, and had no prior cancer. We examined the association between level of eGFR and the risk of incident cancer; the primary outcome was renal cancer, and secondary outcomes were any cancer and specific cancers (urothelial, prostate, breast, lung, and colorectal). During 6,000,420 person-years of follow-up, we identified 76,809 incident cancers in 72,875 subjects. After adjustment for time-updated confounders, lower eGFR (in milliliters per minute per 1.73 m 2 ) was associated with an increased risk of renal cancer (adjusted hazard ratio [HR], 1.39; 95% confidence interval [95% CI], 1.22 to 1.58 for eGFR=45-59; HR, 1.81; 95% CI, 1.51 to 2.17 for eGFR=30-44; HR, 2.28; 95% CI, 1.78 to 2.92 for eGFR,30). We also observed an increased risk of urothelial cancer at eGFR,30 but no significant associations between eGFR and prostate, breast, lung, colorectal, or any cancer overall. In conclusion, reduced eGFR is associated with an independently higher risk of renal and urothelial cancer but not other cancer types.

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“…This index has been shown to be highly specific and unbiased (21,22). Follow-up was for a mean of 3 years for nondiabetic as well as diabetic patients.…”

Section: End Pointsmentioning

confidence: 99%

Effect of Diabetes on Long-Term Mortality Following Contemporary Percutaneous Coronary Intervention

et al. 2004

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OBJECTIVE -Diabetic patients are known to have reduced long-term survival following percutaneous transluminal coronary angioplasty compared with nondiabetic patients. However, it is unknown whether this survival disadvantage has persisted in the era of contemporary percutaneous coronary intervention (PCI) techniques, which include the widespread use of stents and the availability of platelet glycoprotein (GP) IIb/IIIa inhibitors. RESEARCH DESIGN AND METHODS -Three hospitals in New YorkCity contributed prospectively defined data on 4,284 patients undergoing PCI. The primary end point was all-cause mortality following hospital discharge for PCI.RESULTS -Hypertension, renal insufficiency, and renal failure requiring dialysis were all more common in diabetic patients, whereas active smoking was less frequent. Congestive heart failure on admission was more common in diabetic than nondiabetic patients (7.7 vs. 4.0%, P Ͻ 0.001). Stents were placed in 78% of nondiabetic patients and 75% of diabetic patients (P ϭ 0.045). Platelet GP IIb/IIIa antagonists were administered to 23% of nondiabetic and 24% of diabetic patients (P ϭ NS). At a mean follow-up of 3 years, mortality was 8% among nondiabetic patients and 13% for diabetic patients (P Ͻ 0.001). After adjustment for differences in baseline characteristics between nondiabetic and diabetic patients, diabetes remained a significant independent hazard for late mortality (hazard ratio 1.462, 95% CI 1.169 -1.828; P ϭ 0.001).CONCLUSIONS -Following contemporary PCI, diabetic patients continue to have worse survival than nondiabetic patients.

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Use of Commercial Record Linkage Software and Vital Statistics to Identify Patient Deaths

Cited by 189 publications

References 1 publication

Congenital abnormalities and childhood cancer

Congenital abnormalities and childhood cancer

CKD and the Risk of Incident Cancer

Effect of Diabetes on Long-Term Mortality Following Contemporary Percutaneous Coronary Intervention

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