Urticarial Vasculitis

Berg, Robert; Kantor, Gary R.; Bergfeld, Wilma F.

doi:10.1111/j.1365-4362.1988.tb00921.x

Cited by 58 publications

(20 citation statements)

References 36 publications

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“…Nervous system involvement was seen in two patients: one had peripheral neuropathy and the other had seizures. Central nervous system involvement include pseudotumour cerebri, aseptic meningitis and cranial nerve palsies 11–14 . Pseudotumour cerebri, although rare, is the common neurologic complication of UV.…”

Section: Discussionmentioning

confidence: 99%

“…Subsequently, the definition was extended to include patients with a predominantly lymphocytic perivascular infiltrate 15 . It has been suggested that a continuum exists in the histopathologic changes 11 . At one end, a sparse perivascular infiltrate is seen.…”

Section: Discussionmentioning

confidence: 99%

“…Central nervous system involvement include pseudotumour cerebri, aseptic meningitis and cranial nerve palsies. [11][12][13][14] Pseudotumour cerebri, although rare, is the common neurologic complication of UV. None of our patients had cardiac or ocular involvement.…”

Section: Discussionmentioning

confidence: 99%

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Clinicopathologic profile of normocomplementemic and hypocomplementemic urticarial vasculitis: a study from South India

Dincy

George

Jacob

et al. 2008

Acad Dermatol Venereol

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Clinicopathologic profile of normocomplementemic and hypocomplementemic urticarial vasculitis: a study from South India

Dincy

George

Jacob

et al. 2008

Acad Dermatol Venereol

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“…The lesions of urticarial vasculitis with exclusive scalp location are extremely rare [54] . Papular urticaria is the urticarial reaction to an insect bite.…”

Section: Urticarial Lesionsmentioning

confidence: 99%

Scalp Itch: A Systematic Review

Vázquez-Herrera

Sharma

Aleid

et al. 2017

Skin Appendage Disord

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ated with a desire to scratch [1] . Just like skin pruritus, its etiology may be of dermatological or non-dermatological origin. It is common for the dermatologist to encounter patients with no evident cause of scalp pruritus, making it a distressing situation for both the clinician and the patient. The aim of this paper is to review the clinical signs of the most common causes of scalp itch and other less common causes that must be considered in a systematic approach to scalp pruritus. Materials and MethodsWe performed a MEDLINE search through PubMed (1975PubMed ( -2017, using the terms scalp itch and scalp pruritus and included clinical trials, review articles, case series, and case reports to search for the causes of scalp itch. PathophysiologyAlthough various pathogenic etiologies contribute to scalp pruritus, the scalp itself has distinct neuroanatomy and vasculature, specific neuromediators and corresponding receptors, as well as the presence of scalp sebum and microflora, which are all properties that may explain its tendency to be implicated in patients who complain of itch. Keywords Scalp · Itch · Pruritus · Seborrheic dermatitis · Contact dermatitis · Anxiety · Lichen planopilaris · Lice · Pediculosis · Psoriasis · Trichoscopy AbstractScalp itch is a frequent complaint in the dermatological setting. It is common for the dermatologist to encounter patients with no evident cause of scalp pruritus, making it a distressing situation for both the clinician and the patient. The aim of this paper is to propose a systematic approach to scalp itch, which classifies scalp pruritus into two types: (1) with or (2) without dermatological lesions, and presence or absence of hair loss. Also, it is important to think first about the most common causes and then rule out other, less common etiologies. The acronym SCALLP and the five steps for scalp evaluation (listen, look, touch, magnify, and sample) are useful tools to keep in mind for an assertive approach in these patients.

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“…As a second-line drug we propose CyA, ifno contraindications are present. Most authors agree with the dosage of5 mglk/die (28)(29). Ifno adequate control is obtained, alternative immunosuppressive drugs (such as azathioprine or methotrexate) and/or WIG and/or plasmapheresis should be considered (26)(27)(28)(29)(30)(31).…”

Section: Presence Of a Histological Picture Showing Urticarial Vasculmentioning

confidence: 99%

Diagnostic and TherapeuticIterin Chronic Urticaria Patients

Tosoni

Cinquini

2006

Int J Immunopathol Pharmacol

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We describe a diagnostic and therapeutic protocol for the management of chronic urticaria. It is derived from an extensive review of current literature, with a cost-effective evaluation of laboratory investigations and therapeutic approaches. Our protocol may not represent a cornerstone for chronic urticaria: much has in fact to be clarified on pathogenetic mechanisms and aetiological factors. Nevertheless, its application should be able, in our opinion, to identify what is useful or not in the everyday management of chronic urticaria patients.

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Urticarial Vasculitis

Cited by 58 publications

References 36 publications

Clinicopathologic profile of normocomplementemic and hypocomplementemic urticarial vasculitis: a study from South India

Clinicopathologic profile of normocomplementemic and hypocomplementemic urticarial vasculitis: a study from South India

Scalp Itch: A Systematic Review

Diagnostic and TherapeuticIterin Chronic Urticaria Patients

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