Urinary Diversion and Metabolic Acidosis: A Case Report

Abstract

Urinary diversion, a surgical technique to redirect urine usually after cystectomy, and its complications are potential challenges to physicians and early recognition decreases mortality and morbidity. A 45-year-old male with a history of type 2 diabetes mellitus and hypertension for over three years underwent urinary diversion as a treatment for invasive bladder cancer and was discharged ambulatory with an indwelling Foleys catheter. The patient returned to the emergency room with a primary complaint of hematuria. The patient was noted to be frail-looking, dehydrated and tachycardic. Laboratory results yielded an acute kidney injury reflected by the elevated creatinine, hyperkalemia and a venous blood gas demonstrating a hyperchloremic metabolic acidosis. The patient had bilateral distended renal calyces by ultrasound and irrigation of bladder through a three-way Foley showed to have large thick clots. The patient was admitted under the surgical intensive care unit after involving appropriate sub-specialties. The patient was started on sodium bicarbonate infusion, broad-spectrum antibiotics and a urinary catheter in place to monitor urine output. The patient's acidosis steadily improved with correction of his laboratory parameters, transferred out of ICU and the remaining stay in the medical ward was uneventful. The purpose of this case report is to help demonstrate the association between urinary diversion and the type of acidosis that subsequently develops in relation to this surgical procedure.

Hypokalaemic quadriparesis in a patient with urinary diversion through Indiana pouch

“…In a case report by Dsouza and Hussein, a 45-year-old patient who had muscle-invasive bladder carcinoma underwent a urinary diversion procedure in the form of a Studer pouch and was discharged only to be readmitted 3 days after discharge when he presented with haematuria, looking ill, tachycardic and dehydrated 6. He was diagnosed with acute kidney injury and hyperchloraemic metabolic acidosis.…”