Upregulation of TRIM16 mitigates doxorubicin-induced cardiotoxicity by modulating TAK1 and YAP/Nrf2 pathways in mice

Guo, Xinyu; Liu, Mengqing; Han, Bing; Zheng, Yeqing; Zhang, Kaina; Bao, Gaowa; Gao, Chenying; Shi, Hongwen; Sun, Qiang; Zhao, Zhenghang

doi:10.1016/j.bcp.2023.116009

Biochemical Pharmacology

2024

DOI: 10.1016/j.bcp.2023.116009

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Upregulation of TRIM16 mitigates doxorubicin-induced cardiotoxicity by modulating TAK1 and YAP/Nrf2 pathways in mice

Xinyu Guo,

Mengqing Liu,

Bing Han

et al.

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Role of diosmin in preventing doxorubicin-induced cardiac oxidative stress, inflammation, and hypertrophy: A mechanistic approach

AlAsmari,

Al-Shehri,

Algarini

et al. 2024

Saudi Pharmaceutical Journal

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Role of diosmin in preventing doxorubicin-induced cardiac oxidative stress, inflammation, and hypertrophy: A mechanistic approach

AlAsmari,

Al-Shehri,

Algarini

et al. 2024

Saudi Pharmaceutical Journal

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Upregulation of TRIM16 mitigates doxorubicin-induced cardiotoxicity by modulating TAK1 and YAP/Nrf2 pathways in mice

Cited by 1 publication

References 68 publications

Role of diosmin in preventing doxorubicin-induced cardiac oxidative stress, inflammation, and hypertrophy: A mechanistic approach

Role of diosmin in preventing doxorubicin-induced cardiac oxidative stress, inflammation, and hypertrophy: A mechanistic approach

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