Unrecognized Paroxysmal Supraventricular Tachycardia

Lessmeier, Timothy J.; Gamperling, Deborah; Johnson-Liddon, Vicki; Fromm, Barbara S.; Steinman, Russell T.; Meissner, Marc D.; Lehmann, Michael H.

doi:10.1001/archinte.1997.00440260085013

Cited by 117 publications

(13 citation statements)

References 37 publications

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“…Such an abnormality would be expected to magnify sympathetically mediated responses, particularly emotionally driven responses in the heart where noradrenaline inactivation is so dependent on neuronal reuptake (33), perhaps causing sensitization to cardiac symptom development and predisposing to the development of PD. Cardiac patients with palpitations from recurrent supraventricular tachycardias (46) and those with implantable cardiac defibrillators (47), which are prone to discharge in error, certainly are prone to develop PD. Augmentation of the sympathetic neural signal in the heart, by impairment of neuronal noradrenaline reuptake, additionally could increase cardiac risk, predisposing to the development of cardiac tachyarrhythmia (13).…”

Section: Psychophysiology Of Panic Disordermentioning

confidence: 99%

Psychophysiological Mechanisms in Panic Disorder: A Correlative Analysis of Noradrenaline Spillover, Neuronal Noradrenaline Reuptake, Power Spectral Analysis of Heart Rate Variability, and Psychological Variables

Alvarenga

Richards

Lambert

et al. 2006

Psychosomatic Medicine

119

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Section: Psychophysiology Of Panic Disordermentioning

confidence: 99%

Psychophysiological Mechanisms in Panic Disorder: A Correlative Analysis of Noradrenaline Spillover, Neuronal Noradrenaline Reuptake, Power Spectral Analysis of Heart Rate Variability, and Psychological Variables

Alvarenga

Richards

Lambert

et al. 2006

Psychosomatic Medicine

119

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“…The symptoms of paroxysmal SVT may frequently mimic those of panic disorder. In a retrospective survey conducted by Lessmeier et al, 17 women were more likely than men to have their physician attribute their symptoms to panic, anxiety, or stress, thus delaying the correct diagnosis of SVT. Attempting to document rhythm during palpitations helped in determining whether an arrhythmia was truly present.…”

Section: Supraventricular Tachycardiamentioning

confidence: 99%

Gender differences in arrhythmias

Wolbrette

Naccarelli

Curtis

et al. 2002

Clinical Cardiology

100

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Summary: Electrocardiographic and electrophysiologic differences between men and women have long been noted. Women have a higher intrinsic heart rate than men, along with a longer corrected QT interval and a shorter sinus nodal recovery time. The incidence of and risk factors for a variety of arrhythmias differ between men and women. Atrioventricular nodal reentry tachycardia has a 2:1 female-to-male predominance, while accessory pathways are twice as frequent in men. Although atrial fibrillation is more prevalent in men of all age groups, the absolute numbers of men and women with atrial fibrillation are equal, and the associated morbidity and mortality experienced by women with atrial fibrillation appear to be worse. Women have a lower incidence of sudden cardiac death, and female survivors of sudden cardiac death have a lower frequency of spontaneous or inducible ventricular tachycardia. On the other hand, drug-induced torsade de pointes and symptomatic long QT syndrome have a female predominance. Therefore, greater caution should be used when prescribing QT-prolonging drugs in women. The incidence of arrhythmias is increased during pregnancy, and management of pregnant patients poses a significant challenge. The mechanisms of these gender differences are unclear but may be related to hormonal effects and the shorter QT interval in adult males. Pharmacologic and nonpharmacologic therapies are usually equally efficacious, but the risks of pharmacologic therapy are different in men and women. Atrial fibrillation may be more difficult to treat in women.

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“…In some circumstances, patients with primary cardiac arrhythmias can be misdiagnosed as having panic disorder. 27 Accordingly, all patients were clinically assessed by a cardiologist (M.D.E.) participating in the study.…”

Section: Participantsmentioning

confidence: 99%

Sympathetic Activity in Patients With Panic Disorder at Rest, Under Laboratory Mental Stress, and During Panic Attacks

Wilkinson¹,

Thompson²,

Lambert³

et al. 1998

Arch Gen Psychiatry

198

137

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Whole-body and regional sympathetic nervous activity are not elevated at rest in patients with panic disorder. Epinephrine is released from the heart at rest in patients with panic disorder, possibly due to loading of cardiac neuronal stores by uptake from plasma during surges of epinephrine secretion in panic attacks. Contrary to popular belief, the sympathetic nervous system is not globally activated during panic attacks.

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Unrecognized Paroxysmal Supraventricular Tachycardia

Cited by 117 publications

References 37 publications

Psychophysiological Mechanisms in Panic Disorder: A Correlative Analysis of Noradrenaline Spillover, Neuronal Noradrenaline Reuptake, Power Spectral Analysis of Heart Rate Variability, and Psychological Variables

Psychophysiological Mechanisms in Panic Disorder: A Correlative Analysis of Noradrenaline Spillover, Neuronal Noradrenaline Reuptake, Power Spectral Analysis of Heart Rate Variability, and Psychological Variables

Gender differences in arrhythmias

Sympathetic Activity in Patients With Panic Disorder at Rest, Under Laboratory Mental Stress, and During Panic Attacks

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