Unexpected Focal Neurologic Deficit on Emergence From Anesthesia

Oliver, S. B.; Cucchiara, Roy F.; Warner, Mark A.; Muir, Jesse J.

doi:10.1097/00000542-198711000-00039

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Problems Associated With the Trendelenburg Position1

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Cited by 16 publications

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“…One patient had a cerebral hemorrhage during a procedure and emerged with a significant neurological deficit [4]. Lingual and buccal nerve neuropathy can also occur [5].…”

Section: Problems Associated With the Trendelenburg Positionmentioning

confidence: 99%

A brief review: anesthesia for robotic prostatectomy

Baltayian

2008

J Robotic Surg

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“…One patient had a cerebral hemorrhage during a procedure and emerged with a significant neurological deficit [4]. Lingual and buccal nerve neuropathy can also occur [5].…”

Section: Problems Associated With the Trendelenburg Positionmentioning

confidence: 99%

A brief review: anesthesia for robotic prostatectomy

Baltayian

2008

J Robotic Surg

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Perioperative CNS Dysfunction—Diagnosis and Management

Crosby¹

1997

Neuroanesthesia

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Failure to awaken after general anaesthesia secondary to paradoxical venous embolus

Liu¹,

Holley²,

Stulberg³

et al. 1991

Can J Anaesth

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Failure to awaken after general anaesthesia secondary to paradoxical venous embolusProlonged or failed emergence from anaesthesia has been reported from anaesthetic medications, metabolic abnormalities, and neurological causes.t Neurological complications include hypoxia, intracranial haemorrhage, cerebral ischaemia and cerebral embolism. 2 There are several reports of paradoxical air embolism (PAE), causing death or delayed awakening from anaesthesia. 3-5 We report a patient who failed to awaken after an apparently uneventful general anaesthetic, caused by a thrombotic cerebral embolus through a previously unsuspected patent foramen ovale. Case reportA 63-yr-old man presented for revision of a left total hip replacement. He previously had two right and one left hip replacements under general anaesthesia without complications. History revealed no known drug allergies, no abnormal cardio-pulmonary or neurological signs or symptoms, and his only medication was flurbiprofen for arthritis. Non-invasive peripheral vascular impedance plethysmography studies were normal.He received 350 mg thiopentone for induction of anaesthesia, 8 mg pancuronium for muscle relaxation, and 0.5-1.25% isoflurane with 50% nitrous oxide, 250 Ixg fentanyl, and 10 mg morphine for anaesthetic maintenance for an apparently uneventful nine-hour procedure. Intraoperative monitors included ECG, non-invasive sphygmo-manometer, oesophageal thermometer, oesophageal stethoscope, mass spectrometry and peripheral oximeter. The patient was placed in the right lateral position with the head aligned with the torso and not rotated.Blood pressures ranged from 98-140/60-65 mmHg. The pulse rate, which was 60-80 bpm minute during the CAN J ANAESTH 1991 t 38:3 / pp335-7

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Unexpected Focal Neurologic Deficit on Emergence From Anesthesia

Cited by 16 publications

References 0 publications

A brief review: anesthesia for robotic prostatectomy

A brief review: anesthesia for robotic prostatectomy

Perioperative CNS Dysfunction—Diagnosis and Management

Failure to awaken after general anaesthesia secondary to paradoxical venous embolus

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