Ueber das ?Schwielenherz? des S�uglings

Stoeber, E

doi:10.1007/bf00465534

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Clinical Observations (Lncluding Serological Tests With Huma1

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1949

2000

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Cited by 14 publications

(2 citation statements)

References 7 publications

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“…KELLER and VIVELL (1954) who conducted systematic serological studies for three years also noticed a seasonal increase in positive reactions of patients sera during the summer months (Fig. 19) and give (BRENNING 1951;STOEBER 1947STOEBER -1952ÜHIARI 1952) VERLINDE and Fig. 20.…”

Section: Clinical Observations (Lncluding Serological Tests With Humamentioning

confidence: 86%

Columbia SK Group of Viruses

Jungeblut

1958

Handbuch Der Virusforschung

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Section: Clinical Observations (Lncluding Serological Tests With Humamentioning

confidence: 86%

Columbia SK Group of Viruses

Jungeblut

1958

Handbuch Der Virusforschung

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“…HE uidA gene, located at min 36 (BACHMANN 1983), codes for 0-glucu-T ronidase, which is the first enzyme of the hexuronide-hexuronate pathway in Escherichia coli K-12 (ASHWELL 1962;STOEBER 1961). Expression of uidA is principally regulated by the uidR encoded repressor and secondarily by the UxuR repressor (NOVEL and NOVEL 1976).…”

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confidence: 99%

NEGATIVE DOMINANT MUTATIONS OF THE uidR GENE IN ESCHERICHIA COLI: GENETIC PROOF FOR A COOPERATIVE REGULATION OF uidA EXPRESSION

1986

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The uidA gene is the first gene involved in the hexuronide-hexuronate pathway in Escherichia coli K-12 and is under the dual control of the uidR and uxuR encoded repressors. Point mutations affecting the uidR regulatory gene were sought to investigate the regulation of uidA. When the uidR mutant allele was on a multicopy plasmid and the wild-type allele was on the chromosome, some of the mutant phenotypes were dominant to the wild-type phenotype, indicating that the active form of the UidR repressor is multimeric. We have demonstrated that expression of the mutant phenotype is dependent on gene dosage. The dominance of the uidR allele was also sensitive to the presence of the wild-type uxuR allele in the cell. This behavior probably results from UidR-UxuR repressor interactions. A mechanism is proposed: we suggest that the UidR and UxuR repressors interact after their binding to the operator site of uidA; the binding of one regulatory molecule may facilitate the binding of the other one in a cooperative process.

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Dilatative Kardiomyopathie (DCM) Morphologie und Ätiologie. Myokarditis und DCM

Hort¹

2000

Pathologie Des Endokard, Der Kranzarterien Und Des Myokard

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Ueber das ?Schwielenherz? des S�uglings

Cited by 14 publications

References 7 publications

Columbia SK Group of Viruses

Columbia SK Group of Viruses

NEGATIVE DOMINANT MUTATIONS OF THE uidR GENE IN ESCHERICHIA COLI: GENETIC PROOF FOR A COOPERATIVE REGULATION OF uidA EXPRESSION

Dilatative Kardiomyopathie (DCM) Morphologie und Ätiologie. Myokarditis und DCM

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