“…It is generally conceded that antibodies are formed against the virus but the process is probably a very slow one, possibly because the inflammatory reaction provoked by the lesion is very slight and the stimulus to antibody formation may be correspondingly small (Allington, 1952). Thus, Findlay inoculated himself with 3 crops ofwarts before he finally became immune to further inoculations (Findlay, 1930). The speed of antibody formation may well determine to some extent whether a patient will develop one or many lesions, but it does not explain spontaneous resolution since the function of the antibody is to protect normal cells from invasion by the virus while having no effect upon those already invaded, since the virus is intra cellular and antibodies cannot penetrate the intact cell membrane.…”