Treatment of Movement Disorder Emergencies

Frucht, Steven J.

doi:10.1007/s13311-013-0240-3

Cited by 38 publications

(25 citation statements)

References 38 publications

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“…Treatment strategies are variable and depend on the aetiology of OGCs. In druginduced OGCs the first step of management should include removing or, if not possible, reducing the dose of the offending agent [10,119]. In acute cases, administration of anticholinergics, such as benztropine (e.g.…”

Section: Treatmentmentioning

confidence: 99%

“…If response is lacking drug administration should be repeated after 15-30 minutes [119][120][121][122]. To avoid re-occurance of symptoms over the ensuing time frame oral administration of anticholinergics for at least 4-7 days is recommended [119]. Oral administration of anticholinergics may be the most feasible approach for cases seen outside the emergency setting.…”

Section: Treatmentmentioning

confidence: 99%

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Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

Barow

Schneider

Bhatia

et al. 2017

Parkinsonism & Related Disorders

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Oculogyric crisis (OGC) describes the clinical phenomenon of sustained dystonic, conjugate and typically upward deviation of the eyes lasting from seconds to hours. It was initially observed in patients with postencephalitic parkinsonism, but since then a number of conditions have been associated with OGC. These include drug-induced reactions, hereditary and sporadic movement disorders, and focal brain lesions. Here, we systematically review the literature and discuss the spectrum of disorders associated with OGC in order to aid clinicians place this rare but distinctive clinical sign into the appropriate diagnostic context. We also provide a brief synthesis of putative pathophysiological mechanisms, as well as therapeutic recommendations based on the literature and our own experience.

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Section: Treatmentmentioning

confidence: 99%

Section: Treatmentmentioning

confidence: 99%

Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

Barow

Schneider

Bhatia

et al. 2017

Parkinsonism & Related Disorders

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“…Se caracteriza por un cuadro clínico distintivo de cambios en el estado (6) mental, rigidez, fiebre y disautonomía . La mortalidad es resultado de las manifestaciones de las (7) alteraciones autonómicas de la enfermedad y de las complicaciones sistémicas .…”

Section: Las Vías Dopaminérgicasunclassified

Neuroleptic malignant-like síndrome

Vera

Denis

Recalde

et al. 2017

Rev. virtual Soc. Parag. Med. Int

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RESUMENEl síndrome neuroléptico maligno like o crisis acinética es una rara y potencialmente letal complicación del abandono de medicación de los pacientes con enfermedad de Parkinson. Se presenta el caso clínico de un paciente de sexo masculino de 52 años de edad, con antecedente de enfermedad de Parkinson de 10 años de evolución que se encuentra en tratamiento con levodopa-carbidopa. Acude a consultar por disminución del estado de conciencia y rigidez muscular generalizada, relacionado con el abandono de medicación 7 días antes del inicio de los síntomas. Palabras claves: síndrome neuroléptico maligno, enfermedad de Parkinson, receptores dopaminérgicos, dopaminérgicos ABSTRACTNeuroleptic malignant-like syndrome or akinetic crisis is a rare and potentially lethal complication of the withdrawal of medication of patients with Parkinson's disease. We present the clinical case of 52-year man with 10-year history of Parkinson's disease who is being treated with levodopa-carbidopa. He consults for decreased level of consciousness and general muscular rigidity, related to the withdrawal of the medication 7 days before the onset of symptoms. Keywords: neuroleptic malignant syndrome, Parkinson's disease, dopaminergic receptors, dopaminergic drugs INTRODUCCIÓNEl síndrome neuroléptico maligno (SNM) es una complicación rara, pero potencialmente letal, de la utilización de medicamentos antipsicóticos. Es el resultado de una compleja interacción entre la (1) medicación neuroléptica y alguna clase de susceptibilidad . El mecanismo fisiopatológico probablemente se deba al bloqueo de receptores de dopamina a nivel de sistema nervioso central. Esto se ha respaldado en evidencias tales como la asociación con los medicamentos cuyo mecanismo de acción es el bloqueo de receptores dopaminérgicos en el sistema nervioso central, la mayor incidencia entre más fuerte sea la

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“…), Bromocriptin (10-30 mg/24 h, bis 60 mg/24 h) oder Amantadin (200-400 mg/24 h i.v.) eingesetzt werden[30]. Sofern diese Behandlungsstrategien nicht erfolgreich sind, ist eine Elektrokrampftherapie (EKT;[76]) indiziert.Das Serotoninsyndrom kann als unerwünschte Nebenwirkung einer Behandlung mit serotonergen Pharmaka (z.…”

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Pharmakotherapie von psychiatrischen Akut- und Notfallsituationen

Messer¹,

Pajonk

Müller

2015

Nervenarzt

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The pharmacotherapy of psychiatric emergencies is essentially determined by the acuteness, the scene of the emergency, the diagnostic assessment and the special pharmacological profile of the drug used. As there are no specific drugs, syndromic treatment is carried out. For this, primarily antipsychotic drugs and benzodiazepines are available. This article gives an overview of the current state of treatment options for major psychiatric emergency syndromes, namely agitation, delirium, stupor and catatonia, anxiety and panic, as well as drug-induced emergencies.

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Treatment of Movement Disorder Emergencies

Cited by 38 publications

References 38 publications

Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

Oculogyric crises: Etiology, pathophysiology and therapeutic approaches

Neuroleptic malignant-like síndrome

Pharmakotherapie von psychiatrischen Akut- und Notfallsituationen

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