Traumatic renal artery dissection: from imaging to management

An easily misdiagnosed and rare cause of traumatic back pain: bilateral renal infarction caused by traumatic bilateral renal artery dissection

“…Some researchers have suggested the following indications for interventional treatments: (1) hemodynamically stable or stabilized blunt renal injuries; (2) dissection of segmental renal arteries (similar to our case), preocclusive main renal artery dissection, renal artery stenosis without flow limitation, and thrombogenic renal artery intimal tears in imaging patterns on CT; and (3) within 12-24 h after injuries (as early as possible). [7,11] The main known complications of renal artery injury that include renal artery dissection are deterioration of renal function and renovascular hypertension. [7] Among seven reviewed cases with traumatic bilateral renal artery dissection, hypertension occurred in one and renal dysfunction occurred in four cases.…”

Bilateral traumatic renal artery dissection managed by endovascular therapy

“…The widespread use of interventional radiology techniques has made it possible to identify the anatomy of traumatic lesions of the renal arteries. From a radiological point of view, the following types of lesion can be encountered: avulsion of the hilus, dissection of segmental renal arteries, subocclusive dissection of the main renal artery, stenosis of the renal arteries with no limitation of blood flow and thrombogenic lesions of the intima 14. These new contributions to the understanding of this pathology have made it possible to understand the significant disparity in clinical results of revascularisation attempts for a given period of warm ischaemia due to the prognostic importance of the persistence of blood flow to the injured kidney rather than the preprocedural delay 15–17.…”

“…These new contributions to the understanding of this pathology have made it possible to understand the significant disparity in clinical results of revascularisation attempts for a given period of warm ischaemia due to the prognostic importance of the persistence of blood flow to the injured kidney rather than the preprocedural delay 15–17. At the same time, publications reporting a high rate of not only technical (88.9%–100%) but also clinical success (75%–100%) have multiplied and demonstrate an improvement in clinical prognosis of patients suffering from a non-occlusive renal artery lesion limiting blood flow 14 17–19. Covered stents would be indicated in renal vascular lesions.…”

“…To summarise the data that have been presented, surgery would be indicated in the event of uncontrollable life-threatening bleeding, avulsion of the renal pedicle or where there is a progressing retroperitoneal haematoma; non-surgical management could be offered in cases of renal artery occlusion (in the presence of a functional contralateral kidney) or renal artery injury with no limitation of blood flow 14. Finally, endovascular management is a viable option in all other cases as long as the patient’s condition allows it in the trauma context.…”

LncRNA AK154753 promotes acute kidney ischemia-reperfusion injury via modulating the miR-345-3p/Bak and miR-708-5p/Bim axes

“…Acute kidney injury (AKI), also known as acute renal failure, is a clinical syndrome with a rapid decline of renal function caused by multiple factors [1]. The kidney is rich in microvascular network, and the insu cient blood supply is the most crucial factor that leads to renal circulatory disorder and tissue injury [1][2][3]. Furthermore, renal tissue can be damaged again during blood recovery and reperfusion by triggering in ammation or cell apoptosis [4][5][6].…”