“…Left-sided injuries are more common than right (1.4:1-2:1) [2, 3], presumably because the left renal artery is shorter and more acutely angled to the aorta than the right renal artery. The literature describes three mechanisms for secondary thrombosis of renal arteries [2][3][4]11]: a stretch lesion, in which the more elastic components of the arterial wall, adventitia and media, are able to stretch, but the inelastic intima tears, leading to dissection and subsequent thrombosis. Also, contusion of the renal artery against the vertebral column may lead to thrombosis, as well as a retroperitoneal haematoma that compresses the vascular wall.…”