Translocation (1;17) in accelerating and blast phases of chronic myelogeneous leukemia

Kerman, Shelly L.; Miller, Robert B.

doi:10.1016/0165-4608(86)90083-x

Cited by 5 publications

(1 citation statement)

References 20 publications

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“…These were two cases of ALL, one case with inv(11) as the sole karyotypic change 91 and in the second case as part of a complex karyotype, 92 one case of AML M2 in which other chromosome abnormalities were present, 93 one of CMML 94 and one case of CML. 95 One case of AML M6 was reported with a complex rearrangement of chromosome 11 in addition to the inversion, der(11)inv(11)(p15q23) del(11)(p12p13). 96 One translocation t(11;11)(p15;q23) has been previously described in a case of AML M5 97 and one ring 11, r(11)(p15q23), in a case of AML M6 with a complex karyotype.…”

Section: (C) Chromosome Inversionsmentioning

confidence: 99%

Ten novel 11q23 chromosomal partner sites

Cuneo²,

et al. 1998

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The MLL gene located at 11q23 has been described as a 'promiscuous' gene due its involvement with a large number of genetic partners. The EU Concerted Action Workshop on 11q23 provided 550 cases for study of which 82 showed abnormalities which did not involve the established translocations or deletion of 11q23. In these 'other' cases, which included inversions and duplications, 11q23 was found to be involved with 25 chromosome partners of which 10 had not been previously reported. These were 1q31, 4p11, 6q13, 8q21, 10q22, 10q25, 11q11, 11q21, 13q34 and 18q23. This study demonstrated the value of the Workshop, in confirming the diversity of chromosomal partner sites involved with 11q23 and in the identification of new partners.

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