“…Cardiac tamponade is a medical emergency that restricts the filling of the heart chambers and alters normal hemodynamic conditions, resulting in cardiogenic shock and hypotension. It happens after a quick or overflow of fluid into the pericardium cavity [1].…”
Pericardial decompression syndrome (PDS) is an unusual, potentially fatal complication that occurs after pericardial drainage for cardiac tamponade. either by needle pericardiocentesis or surgical pericardiostomy. It manifests with paradoxical hemodynamic deterioration and/or pulmonary edema, commonly associated with ventricular dysfunction. PDS usually begins after initial clinical amelioration after pericardiocentesis. It is largely under-reported and may be neglected in clinical practice. While the precise mechanisms behind PDS are not well understood, it seems to be strongly related to patients with preexisting ventricular dysfunction. Doctors who perform pericardial drainage should be mindful of the associated high-risk factors for the intervention, taking into consideration the uncommon possibility of PDS formation.
“…Cardiac tamponade is a medical emergency that restricts the filling of the heart chambers and alters normal hemodynamic conditions, resulting in cardiogenic shock and hypotension. It happens after a quick or overflow of fluid into the pericardium cavity [1].…”
Pericardial decompression syndrome (PDS) is an unusual, potentially fatal complication that occurs after pericardial drainage for cardiac tamponade. either by needle pericardiocentesis or surgical pericardiostomy. It manifests with paradoxical hemodynamic deterioration and/or pulmonary edema, commonly associated with ventricular dysfunction. PDS usually begins after initial clinical amelioration after pericardiocentesis. It is largely under-reported and may be neglected in clinical practice. While the precise mechanisms behind PDS are not well understood, it seems to be strongly related to patients with preexisting ventricular dysfunction. Doctors who perform pericardial drainage should be mindful of the associated high-risk factors for the intervention, taking into consideration the uncommon possibility of PDS formation.
“…The most prevalent pathophysiologic hypothesis states that sudden increase in ventricular preload with concomitantly increased afterload (sympathetic drive) leads to TVD. [1][2][3] We present a case of asymptomatic TVD with classical features of SCM after PCC for imminent cardiac tamponade. case report: 49-year-old female presented with 1 month history of chest pain, dyspnea and dry cough.…”
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