Training Level Comparison Study: EFFECT OF HIGH AND LOW INTENSITY EXERCISE ON VENTILATORY THRESHOLD IN MEN WITH CORONARY ARTERY DISEASE

Jensen, Betsy E.; Fletcher, Barbara J.; Rupp, Jeffrey C.; Fletcher, Gerald F.; Lee, Jeanette Y.; Oberman, Albert

doi:10.1097/00008483-199607000-00003

Cited by 31 publications

(29 citation statements)

References 13 publications

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“…Numerous studies have shown that aerobic exercise training (ET) at or above the LT and VT results in a greater oxygen consumption (V'O 2 ) at which they occur. This has been noted both in healthy subjects and individuals with CAD (Jensen et al 1996;Jones and Carter 2000;Poole and Gaesser 1985). Whether ET elicits a similar training response in the DPBP has not yet been investigated.…”

Section: Introductionmentioning

confidence: 93%

The influence of aerobic exercise training on the double product break point in low-to-moderate risk adults

et al. 2010

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The double product (DP) is the product of the heart rate (HR) and systolic blood pressure (SBP). The double product break point (DPBP) is a physiologic threshold that occurs at similar exercise intensities to that of the ventilatory threshold (VT). The influence of aerobic exercise training on the DPBP has not yet been examined. The purpose of this study was to examine whether aerobic exercise training (ET) increases the exercise intensity at which the DPBP occurs, and whether it increases in a similar fashion to the VT. Seven males and 11 females, all sedentary (mean ± SD: age = 29.9 ± 10.5 yr) underwent supervised cardiopulmonary exercise testing using a cycle ergometer ramp protocol at baseline and after eight weeks of vigorous ET on a cycle ergometer. The VT was determined by gas analysis and the V-slope method. Experienced observers using standardized instructions visually determined the DPBP. Following ET, VO 2peak , maximal workload, and body composition variables all showed significant positive changes. The VO 2 at which the DPBP and VT occurred increased significantly from baseline to follow-up (p <.001). At baseline and at follow-up, the DPBP and VT did not differ. The DPBP and VT were significantly correlated to each other at both time points. Results suggest that the DPBP responds to ET in a similar fashion to that of the VT, and may be an easier and more useful marker of the VT for exercise training purposes.

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Section: Introductionmentioning

confidence: 93%

The influence of aerobic exercise training on the double product break point in low-to-moderate risk adults

et al. 2010

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“…Although defining studies of healthy individuals are still lacking, clinical trials point to the superiority of high aerobic exercise intensity over low‐to‐moderate intensities to gain full effect of an exercise training programme (Shephard 1968, Jensen et al. 1996, Tanasescu et al.…”

Section: Exercise Intensitymentioning

confidence: 99%

Mechanisms of exercise‐induced improvements in the contractile apparatus of the mammalian myocardium

Kemi

Wisløff

2010

Acta Physiologica

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One of the main outcomes of aerobic endurance exercise training is the improved maximal oxygen uptake, and this is pivotal to the improved work capacity that follows the exercise training. Improved maximal oxygen uptake in turn is at least partly achieved because exercise training increases the ability of the myocardium to produce a greater cardiac output. In healthy subjects, this has been demonstrated repeatedly over many decades. It has recently emerged that this scenario may also be true under conditions of an initial myocardial dysfunction. For instance, myocardial improvements may still be observed after exercise training in post-myocardial infarction heart failure. In both health and disease, it is the changes that occur in the individual cardiomyocytes with respect to their ability to contract that by large drive the exercise training-induced adaptation to the heart. Here, we review the evidence and the mechanisms by which exercise training induces beneficial changes in the mammalian myocardium, as obtained by means of experimental and clinical studies, and we argue that these changes ultimately alter the function of the whole heart and contribute to the changes in whole-body function.

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“…Concerning intensity, the same Position Stand [5] recommends exercise at 55 to 90% of the maximal heart rate, which corresponds to 40 to 85% of maximal aerobic power. Within this range, one study [22] found greater improvements in patients with CAD who trained at a higher intensity (85%) compared with patients who trained at a lower intensity (50%).…”

Section: Determinantsmentioning

confidence: 99%

Determinants of the effects of physical training and of the complications requiring resuscitation during exercise in patients with cardiovascular disease

Vanhees

Stevens

Schepers

et al. 2004

European Journal of Cardiovascular Prevention & Rehabilitat

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Training Level Comparison Study: EFFECT OF HIGH AND LOW INTENSITY EXERCISE ON VENTILATORY THRESHOLD IN MEN WITH CORONARY ARTERY DISEASE

Abstract: Among this population, both HI and LO improve the VO2 at VT and peak, but the improvement is greater with HI. The VT can be a useful adjunct to heart rate and peak oxygen uptake when prescribing exercise.

Cited by 31 publications

References 13 publications

The influence of aerobic exercise training on the double product break point in low-to-moderate risk adults

The influence of aerobic exercise training on the double product break point in low-to-moderate risk adults

Mechanisms of exercise‐induced improvements in the contractile apparatus of the mammalian myocardium

Determinants of the effects of physical training and of the complications requiring resuscitation during exercise in patients with cardiovascular disease

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