2016
DOI: 10.1016/j.ijpam.2015.12.001
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Torsion of wandering spleen

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Cited by 2 publications
(7 citation statements)
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“…The pathophysiology of WS is characterised by the hypermobility of the spleen secondary to the absence or excessive laxity of the following primary splenic suspensory ligaments: gastrosplenic, splenorenal, splenocolic, splenophrenic, pancreaticosplenic, and pre-splenic folds [1,4,[6], [7], [8], [9], [10],13,16]. Due to the abnormality or absence of these ligaments, the splenic vascular pedicle in WS is susceptible to elongation and torsion [1,7,8,12]. The aetiology of WS is considered to be multifactorial and broadly divided into congenital anomalies and acquired conditions [1,6,7].…”
Section: Discussionmentioning
confidence: 99%
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“…The pathophysiology of WS is characterised by the hypermobility of the spleen secondary to the absence or excessive laxity of the following primary splenic suspensory ligaments: gastrosplenic, splenorenal, splenocolic, splenophrenic, pancreaticosplenic, and pre-splenic folds [1,4,[6], [7], [8], [9], [10],13,16]. Due to the abnormality or absence of these ligaments, the splenic vascular pedicle in WS is susceptible to elongation and torsion [1,7,8,12]. The aetiology of WS is considered to be multifactorial and broadly divided into congenital anomalies and acquired conditions [1,6,7].…”
Section: Discussionmentioning
confidence: 99%
“…Most patients with WS are asymptomatic [10]. Commonly, WS is detected incidentally in physical examination as an abdominal mass or in imaging studies performed for other conditions [3,[6], [7], [8],[12], [13], [14]]. Paediatric WS patients usually present with acute abdominal pain, whereas abdominal mass is the most common complaint in adult WS patients [6].…”
Section: Discussionmentioning
confidence: 99%
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