Tinea Capitis Treated with Griseofulvin

Stritzler, Conrad; Rein, Robert L.; Ulku, Kemal

doi:10.1001/archderm.1962.01590060053011

Cited by 5 publications

(2 citation statements)

References 9 publications

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“…Although griseofulvin has been used as intermittent therapy 13–26 in tinea capitis, the preferred regimen is the continuous mode. 10–11 In one study, fluorescent Microsporum tinea capitis was treated with griseofulvin plus benzoic and salicylic acid ointment.…”

Section: Griseofulvinmentioning

confidence: 99%

“…Itraconazole: an alternative regimen is continuous therapy 5 mg/kg/day × 2–4 weeks. 10–69 The number of pulses required for tinea capitis is in part dependent upon the baseline severity of tinea capitis and the causative organism. Terbinafine: the dosage regimen for: weight < 20 kg, &frac14 tablet/day; 20–40 kg, ½ tablet/day; and > 40 kg, 1 tablet (250 mg)/day.…”

Section: Itraconazolementioning

confidence: 99%

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An evaluation of intermittent therapies used to treat onychomycosis and other dermatomycoses with the oral antifungal agents

Gupta

Rosso²

2000

Int J Dermatology

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Section: Griseofulvinmentioning

confidence: 99%

Section: Itraconazolementioning

confidence: 99%

An evaluation of intermittent therapies used to treat onychomycosis and other dermatomycoses with the oral antifungal agents

Gupta

Rosso²

2000

Int J Dermatology

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Literatur

Wenzel¹,

Gudenau²,

Moeljono³

1977

Sinterung Von Filterkuchen Aus Eisenerz- Bzw. Konzentratschlamm

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Tinea capitis

2000

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Tinea capitis is a common dermatophyte infection of the scalp in children. Dermatophytes are classified into three genera; tinea capitis is caused predominantly by Trichophyton or Microsporum species. On the basis of host preference and natural habitat, dermatophytes are also classified as anthropophilic, geophilic and zoophilic. The etiological agents of tinea capitis usually fall in the first and last categories. In North America, tinea capitis is now predominantly due to Trichophyton tonsurans. During the past 100 years the most common North American organism for tinea capitis was initially Microsporum canis followed later by M. audouinii. In other parts of the world the epidemiology varies. Tinea capitis is generally observed in children over the age of 6 years and before puberty, with African Americans being the most affected group. Clinical presentations are seborrheic-like scale, 'black dot' pattern, inflammatory tinea capitis with kerion and tiny pustules in the scalp. The clinical diagnosis should be confirmed by mycological examination. Wood's light examination was of value in diagnosing tinea capitis due to M. canis and M. audouinii; however, it is not helpful in T. tonsurans tinea capitis. Asymptomatic carriers may be a significant reservoir of infection and spread of spores may also involve inanimate objects. Carriers may benefit from shampooing their hair. Treatment of tinea capitis requires an oral antifungal agent. The data from the use of terbinafine, itraconazole and fluconazole are promising and suggest that these agents have an efficacy similar to griseofulvin while shortening the duration of therapy. Both griseofulvin and the newer antimycotics have a favorable adverse-effect profile and are associated with high compliance.

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Tinea Capitis Treated with Griseofulvin

Cited by 5 publications

References 9 publications

An evaluation of intermittent therapies used to treat onychomycosis and other dermatomycoses with the oral antifungal agents

An evaluation of intermittent therapies used to treat onychomycosis and other dermatomycoses with the oral antifungal agents

Literatur

Tinea capitis

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