Time Course of Intraocular Hypertension Produced by Suxamethonium

Intraocular Tension Variations With Electroconvulsive Therapy

“…This finding is contrary to Kalinowsky [3J but is in agreement with few other studies [4,51. On the other hand in modified ECT even before the convulsion, succinylcholine itself elevated the lOT which is a known effect of the drug [7,8] MJAFI, 50 : 4, OCTOBER 1994 and is in agreement with the findings of Shukla et aIlS). The present study showed that as compared to direct ECT the rise of lOT after modified ECT was much marc pronounced and significantly longer lasting due to the effect of succinylcholine.…”

“…The mean age of the patients was 28.3 years. The commonest diagnosis was schizophrenia (67.5%) followed 7) which were continued during the course of ECT but in reduced dosages.…”

The effect of propofol/remifentanil rapid-induction technique without muscle relaxants on intraocular pressure

“…Remifentanil rapidly caused a decrease in IOP after induction, which remained unaffected by the stimulus of laryngoscopy and intubation. Succinylcholine produces an increase in IOP beginning at one minute after its administration, peaks within two to 4 minutes [15,16], and this IOP increase is exaggerated by laryngoscopy and intubation [17]. The patients receiving succinylcholine had a slight decrease in IOP immediately after induction at Time-1.…”

Anesthetic management for a patient with oculocerebrorenal (Lowe's) syndrome

“…An increase in IOP due to hypercapnea or hypoxia appears to be associated with changes in intracranial pressure and with retinal vasodilation [12]. The IOP is also increased by succinylcholine [13] and endotracheal intubation especially when it is difficult or prolonged [14]. Preventing fasciculations by pretreatment with a small dose of a nondepolarizing muscle relaxant may attenuate the IOP response to succinylcholine without causing obvious eye damage [15].…”