Thrombosis of Inferior Cerebellar Artery

Menzies, W. F.

doi:10.1093/brain/16.3.436

Cited by 8 publications

(2 citation statements)

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“…80 The first case reports of surgical treatment by craniectomy were published independently but almost simultaneously in 1956 by Fairburn and Olivier 26 and Lindgren. 73 As of the writing of this study, 115 original papers on space-occupying cerebellar stroke including a total of over 750 patients have been published (see Table 3).…”

Section: Neurosurg Focus 34 (5):e8 2013mentioning

confidence: 99%

Space-occupying cerebellar infarction: complications, treatment, and outcome

Neugebauer

Witsch

Zweckberger

et al. 2013

FOC

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Space-occupying brain edema is a frequent and one of the most dreaded complications in ischemic cerebellar stroke. Because the tight posterior fossa provides little compensating space, any space-occupying lesion can lead to life-threatening complications through brainstem compression or compression of the fourth ventricle and subsequent hydrocephalus, both of which may portend transtentorial/transforaminal herniation. Patients with large cerebellar infarcts should be treated and monitored very early on in an experienced stroke unit or (neuro)intensive care unit. The general treatment of ischemic cerebellar infarction does not differ from that of supratentorial ischemic strokes. Treatment strategies for space-occupying edema include pharmacological antiedema and intracranial pressure–lowering therapies, ventricular drainage by means of an extraventricular drain, and suboccipital decompressive surgery, with or without resection of necrotic tissue. Timely escalation of treatment is crucial and should be guided by clinical and neuroradiological rationales. Patients in a coma after hydrocephalus and/or local brainstem compression may also benefit from more aggressive surgical treatment, as long as the conditions are reversible. Contrary to the general belief that outcome in survivors of space-occupying cerebellar stroke is usually good, recent studies suggest that for many of these patients, the long-term outcome is not good. In particular, advanced age and additional brainstem infarction seem to be predictors for poor outcome. Further trials are necessary to investigate these findings systematically and provide better selection criteria to help guide decisions about surgical therapies, which should always be carried out in close cooperation among neurointensive care physicians, neurologists, and neurosurgeons.

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Section: Neurosurg Focus 34 (5):e8 2013mentioning

confidence: 99%

Space-occupying cerebellar infarction: complications, treatment, and outcome

Neugebauer

Witsch

Zweckberger

et al. 2013

FOC

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“…A few detailed clinicopathologic reports based on single cases or short series are available. 1 - 9 In a larger series of 28 acute massive cerebellar infarcts Sypert and Alvord 10 found 31 arterial occlusions. The most common site of occlusion (18 of 31) was the distal vertebral artery (VA), but causes of the occlusions were not specified.…”

mentioning

confidence: 97%

Arterial pathology in cerebellar infarction.

Amarenco

Hauw

Gautier

1990

Stroke

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We reviewed 88 pathologically proven cerebellar infarcts, examining the entire thoracocervical arterial system (including the spinal part of the vertebral artery, the intracranial arteries, and the heart) in all cases. For 50 infarcts, we found an arterial occlusion. The intracranial part of the vertebral artery was involved in 38 infarcts (76%), the basilar artery in 20 infarcts (40%), and a cerebellar artery in 12 infarcts (24%). Eight infarcts were due to bilateral distal vertebral artery occlusions. For the 50 infarcts, the cause of the occlusion was atherosclerosis in 28 (56%) and a cardiac embolism in 12 (24%). For the remaining 38 infarcts, we found no arterial occlusions on postmortem examination. Of these 38 infarcts, 26 (68%) were associated with a cardiac source of emboli. For the entire group of 88 infarcts, atherosclerosis was the cause in 31 (35%), while a cardiac embolism was proven or presumed in 38 (43%). Dissecting aneurysms, atherosclerotic artery-to-artery embolism, or hemodynamic mechanisms could have been responsible for a few infarcts. We conclude that cerebellar infarcts often arise from cardiogenic embolism. (Stroke 1990;21:1299-1305)

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German-Austrian Scape Occupying Cerebellar Infarction Study (GASCIS): study design, methods, patient characteristics

Krieger¹,

Busse

Schramm³

et al. 1992

J Neurol

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An open prospective multicentric therapeutic trial to determine the timing and type of therapeutic intervention in patients presenting with secondary deterioration following cerebellar stroke is described. According to the results of retrospective studies a controlled approach comparing different therapies is ethically not feasible. Participants use the same scores and protocol for patient data collection but many choose different therapeutic procedures.

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Thrombosis of Inferior Cerebellar Artery

Cited by 8 publications

References 0 publications

Space-occupying cerebellar infarction: complications, treatment, and outcome

Space-occupying cerebellar infarction: complications, treatment, and outcome

Arterial pathology in cerebellar infarction.

German-Austrian Scape Occupying Cerebellar Infarction Study (GASCIS): study design, methods, patient characteristics

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