Thermoregulation and Plasma Cateoholamines During Submaximal Work in Individuals Susceptible to Malignant-Hyperpyrexla

Ayling, Jacqui; Ellis, F. R.; Halsall, P.J.; Campbell, Iain; Qrrle, S; Caddy, Josh

doi:10.1249/00005768-198504000-00414

Medicine & Science in Sports & Exercise

1985

DOI: 10.1249/00005768-198504000-00414

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Thermoregulation and Plasma Cateoholamines During Submaximal Work in Individuals Susceptible to Malignant-Hyperpyrexla

Jacqui Ayling

F. R. Ellis

P.J. Halsall

et al.

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1995

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Neuroleptic malignant syndrome and malignant hyperthermia: end of a controversy?

1995

JNP

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Two primary hypotheses have been proposed to explain the pathophysiology of the neuroleptic malignant syndrome (NMS): 1) that NMS is produced by abrupt and extensive central dopamine receptor blockade by neuroleptics, particularly in nigrostriatal and hypothalamic pathways; and 2) that NMS, like malignant hyperthermia (MH), results from a preexisting defect in skeletal muscle metabolism that is unmasked or provoked by neuroleptic exposure. To evaluate these models, the authors review studies published since 1980 of the clinical features, epidemiology, risk factors, laboratory assessment, and relevant animal models of NMS and MH. Data from these studies suggest that although NMS and MH are clinically similar, they are pharmacologically distinct, implying that cross-reactivity between triggering agents is unlikely to occur.

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Neuroleptic malignant syndrome and malignant hyperthermia: end of a controversy?

1995

JNP

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Thermoregulation and Plasma Cateoholamines During Submaximal Work in Individuals Susceptible to Malignant-Hyperpyrexla

Cited by 1 publication

References 0 publications

Neuroleptic malignant syndrome and malignant hyperthermia: end of a controversy?

Neuroleptic malignant syndrome and malignant hyperthermia: end of a controversy?

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