The synthesis of β-adrenergic receptors in cultured human lung cells: Induction by glucocorticoids

In Vitro Desensitization of Beta Adrenergic Receptors in Human Neutrophils. ATTENUATION BY CORTICOSTEROIDS

“…Steroid hormones increase beta adrenergic receptor density in human (14,31,32) and animal (33,34) tissues. Further, steroid hormones enhance adenylate cyclase action (assessed either by enzyme activity or cAMP accumulation) (14,28,(35)(36)(37).…”

Effects of fasting on hepatic catecholamine receptors

“…fl-Adrenergic agonists and antagonists competed with []25I]CYp binding in a manner consistent with its binding to fl-adrenergic receptors. Binding of CYP was also stereospecific, with [18]. This increase in fi-receptor sites represents a functional increase with respect to fi-adrenergic action as shown by an enhanced epinephrine-sensitive cyclic AMP accumulation in hepatocytes isolated from fasted rats (table 2).…”

Dexamethasone‐dependent expression of β1–24 corticotropin stimulated adenylate cyclase during adipose conversion of 3T3‐F442A cells

“…Otherwise glucocorticoid hormones have been shown to have physiological actions on multiple cellular processes; thus, the indirect, so-called 'permissive' actions may involve the modulation of the affinity and/or receptor concentrations of a variety ot ~ heterologous hormone and growth factor receptors [28][29][30][31][32][33][34][35][36][37][38][39][40]. So, given the data presented in this paper, ACTH could regulate its own receptor in adipocytes early during adipose tissue formation via glucocorticoid secretion from the adrenal gland; thus a dual effector mechanism of ACTH action should be retained to render an account of the biological response of the adipose cell to this hormone.…”