1969
DOI: 10.1093/brain/92.4.743
|View full text |Cite
|
Sign up to set email alerts
|

The Syndrome of Primary Orthostatic Hypotension

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2
1
1

Citation Types

3
22
1

Year Published

1971
1971
1995
1995

Publication Types

Select...
9

Relationship

1
8

Authors

Journals

citations
Cited by 64 publications
(26 citation statements)
references
References 0 publications
3
22
1
Order By: Relevance
“…in patients with orthostatic hypotension due to other neurological disorders have produced conflicting evidence for the role of the sym pathetic nervous system in renin release. It has been suggested that an integrated sympathetic nervous system is necessary (Gordon, Kuchel, Liddle & Island, 1967;Chokroverty, Barron, Katz, Del Greco & Sharp, 1969) but our present observations fail to support this view. Corbett, Frankel and Harris (1971C) have investigated the cardiovascular responses to change of posture in tetraplegic man and considered that any blood pressure overshoot they observed immediately after return to the supine position was associated with a spasm of skeletal muscles.…”
contrasting
confidence: 99%
“…in patients with orthostatic hypotension due to other neurological disorders have produced conflicting evidence for the role of the sym pathetic nervous system in renin release. It has been suggested that an integrated sympathetic nervous system is necessary (Gordon, Kuchel, Liddle & Island, 1967;Chokroverty, Barron, Katz, Del Greco & Sharp, 1969) but our present observations fail to support this view. Corbett, Frankel and Harris (1971C) have investigated the cardiovascular responses to change of posture in tetraplegic man and considered that any blood pressure overshoot they observed immediately after return to the supine position was associated with a spasm of skeletal muscles.…”
contrasting
confidence: 99%
“…Clark et al (1979) reported that diabetic neuropathy was incapable to increase norepinephrine and renin release in response to postural changes with impaired sympathetic efferent nerve, which might, in turn, result in hypotension. Bannister et al (1977) and Chokroverty et al (1969) also showed the similar findings in non-diabetic neurophathy. In the present study, plasma norepinephrine and renin activity were not determined,but observed postural hypotension might indicate the presence of impaired catecholamine and renin release.…”
Section: Discussionsupporting
confidence: 77%
“…We also found ten subjects with EMG 17 workers studied (40%) changes in the EMG that were indicative of CTS.7 Nevertheless, his data contain no clinical evaluation and it must be emphasised that a diagnosis of CTS must be based on clinical verification. Ahlborg and Voog undertook a case control study of the occurrence of CTS among workers exposed to hand-arm vibration and according to their results, heavy manual work increased the risk of CTS by a factor of two; when combined with handarm vibration the risk was increased by a factor offive.…”
Section: Discussionmentioning
confidence: 76%