The surgical significance of the blood supply of the canine stifle joint

Abstract

The blood supply of the stifle joint was investigated in ten fresh dog cadavers using injections of Micropaque into the aorta and subsequent radiographic examination. The relevance of the blood supply to surgical intervention and the repair of injuries is discussed.

Histologic Changes in Ruptured Canine Cranial Cruciate Ligament

“…Furthermore, it is also unclear whether loss of these cells occurs through necrosis or apoptosis. Development of progressive tissue hypoxia over time may be an important factor mediating this loss of cells because the CCL has a relatively tenuous microvasculature, which arises predominantly from the epiligamentous tissue rather than the proximal and distal bony attachment sites 14,15 …”

Ex Vivo Evaluation of the Effect of Tibial Plateau Osteotomy on the Proximal Tibial Soft Tissue Envelope With and Without the Use of Protective Gauze Sponges

“…Preservation of the caudal periosteal arterioles that penetrate the femur via the adductor magnus et brevis muscle is recognized as being important during open reduction and fixation of femoral diaphyseal fractures in which the medullary blood supply has been severed. 24 A similar scenario occurs during TPLO in which the major source of intraosseous arterial supply to the proximal tibia (a branch of the popliteal artery which penetrates the tibia via a nutrient foramen located distolateral to the osteotomy) 25 is severed by the oscillating saw. Vascular compromise secondary to soft tissue dissection has also been implicated as a predisposing factor for tibial tuberosity fracture, which is intermittently reported as a complication of TPLO.…”

Thermal Imaging of Normal and Cranial Cruciate Ligament‐Deficient Stifles in Dogs

“…The high incidence of CCL failure suggests premature ligament degeneration as an underlying cause. Predisposing factors include aging (especially large breed dogs), conformational abnormalities (straight pelvic limbs), immune‐mediated disorders, genetic predisposition, excessive biomechanical stress, and decreased blood supply to the center of the ligament 2–5 …”