1948
DOI: 10.1080/00325481.1948.11692999
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The Shoulder-Hand Syndrome in Reflex Dystrophy of the Upper Extremity

Abstract: was noted, although the patient did not complain of pain. At this time, there was some atrophy of the intrinsic muscles of the hands. Soon stiffness of the fingers was noted, followed by flexion contractures. Sensory examination now revealed areas of hypesthesia in the hands and forearms. The skin of the hands was cool and showed trophic changes and patchy areas of cyanosis.Case 3.

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Cited by 21 publications
(18 citation statements)
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“…The 'warm' and 'cold' forms represent another classification of CRPS based on the main clinical features. This is not a formal classification and was primarily described by Steinbrocker at the end of the 50s (51). The clinical presentation of the distal limb permits to distinguish the two forms (red, and edematous extremity in the warm form and dusky, sweaty extremity in the cold one).…”
Section: Clinical Features and Subtypesmentioning
confidence: 99%
“…The 'warm' and 'cold' forms represent another classification of CRPS based on the main clinical features. This is not a formal classification and was primarily described by Steinbrocker at the end of the 50s (51). The clinical presentation of the distal limb permits to distinguish the two forms (red, and edematous extremity in the warm form and dusky, sweaty extremity in the cold one).…”
Section: Clinical Features and Subtypesmentioning
confidence: 99%
“…Peripheral vasoconstriction has been held to be responsible for referred cardiac pain by Roberts (1945) and has been demonstrated by variations of cutaneous temperature (Doret and Ferrero, 1951). An analogous mechanism is known to be responsible for the trophic lesions in the upper limb which are well-recognized clinical sequelae of acute cardiac pain and myocardial infarction (Askey, 1941;Kehl, 1943;Johnson, 1943;Steinbrocker, Spitzer, and Friedman, 1948;Hilker, 1949;Chitwood, 1950;Swan and Henderson, 1951). Apart from possibly initiating reflexes, which may cause spasm of the peripheral vessels, the coronary arteries themselves are subject to vasospastic stimuli from various organs of the body (von Bergmann, 1932;Greene, 1935;Gilbert, Fenn, and LeRoy, 1940;Freedberg, Spiegi, and Riseman, 1944).…”
Section: Discussionmentioning
confidence: 99%
“…Increased atmospheric pressure is another hazard, and McCallum, Stanger, Walder, and Paton (1954) associated this with bilateral avascular necrosis of the femoral heads. On the other hand, some diseases, apparently related to the musculoskeletal system, have no direct traumatic background; for instance, the shoulder-hand syndrome may co-exist with a coronary infarct (Steinbrocker, Spitzer, and Friedman, 1948).…”
Section: Severity Of Rheumatic Diseasementioning
confidence: 99%