“…Most now consider the disorder to be biologically determined (Rutter, 1988) with evidence implicating genetic factors (Bolton & Rutter, 1990), but also indicating weaker associations with birth complications (Tsia, 1987), minor congenital anomalies (Campbell, Celler, Small, Petti & Ferris, 1978) and epilepsy (Deykin & MacMahon, 1979a;Rutter, 1970). In addition, pre-, peri-and postnatal infections (rubella, herpes simplex, cytomegalovirus) have been found to co-occur (Coleman, 1987;Deykin & MacMahon, 1979b;Knobloch & Pasamanick, 1975). In view of this evidence, there are several hypotheses that may account for seasonal effects in autism and each leads to a rather different set of expectations.…”