The risk of adenoid hypertrophy in children with allergic rhinitis

Huang, Shih-Wen; Giannoni, Carla M.

doi:10.1016/s1081-1206(10)62251-x

Cited by 96 publications

(68 citation statements)

References 16 publications

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“…Many triggers, including microbial stimuli such as molds (2119) or external irritants like cigarette smoke (2120), have been related to the enlargement of adenoid tissue and hence to the development of symptoms. Symptoms related to adenoid hypertrophy range from nasal obstruction, rhinolalia clausa, open-mouth breathing and snoring to the so-called Ôadenoid faciesÕ.…”

Section: Adenoid Hypertrophymentioning

confidence: 99%

Allergic Rhinitis and its Impact on Asthma (ARIA) 2008*

Bousquet¹,

Khaltaev²,

Cruz³

et al. 2008

Allergy

3,754

1,761

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Section: Adenoid Hypertrophymentioning

confidence: 99%

Allergic Rhinitis and its Impact on Asthma (ARIA) 2008*

Bousquet¹,

Khaltaev²,

Cruz³

et al. 2008

Allergy

3,754

1,761

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“…Eight patients (13.33%) had adenoid hypertrophy on diagnostic nasal endoscopy. In a study by Huang and Giannoni [21] on the risk of adenoid hypertrophy in children with AR, there was a significant association between the two. In addition, 8 patients (13.33%) had signs of sinusitis on endoscopy.…”

Section: Bellamkonda and Vinodkumarmentioning

confidence: 95%

A Clinicopathological Study of Allergic Rhinitis

Bellamkonda

Kumar

2016

Asian J Pharm Clin Res

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Objectives: Allergic rhinitis (AR) is on the rise in our society for various reasons. It is said that allergy might represent a primary or secondary factor in up to half the patients encountered in an otolaryngology practice. Thus, the management of AR constitutes a large proportion of the day-to-day practice. In addition to its primary effect, inhalant allergy of the upper respiratory tract might affect the development and clinical course of other disease states such as sinusitis, otitis media, and asthma due to the mucosal continuity. Hence, this study was conducted to know the comorbid associations of AR and also the pathology in inferior turbinate hypertrophy (ITH). Methods:A time-bound, descriptive cross-sectional study was conducted in ENT OPD. Patients were selected for the study after proper history taking, clinical examination, laboratory investigations such as absolute eosinophil count (AEC) and diagnostic nasal endoscopy, and consent for biopsy from inferior turbinate and histopathological examination using predefined inclusion and exclusion criteria. Nasal symptoms of sneezing, rhinorrhea, nasal pruritus, nasal obstruction, anosmia and non-nasal symptoms of eye itching, watering, foreign body sensation, and pharyngeal itching were recorded. Results:The mean age of the patients was 27.08 years with a standard deviation of 6.1. Patients were clinically evaluated regarding symptoms in ear, nose, throat, eyes, and respiratory system. All patients had nasal symptoms whereas 53.34% patients had ophthalmic and pharyngeal symptoms. Aural comorbidity was most common with complaints in 68.33% followed by respiratory symptoms in 30% patients. About 40% patients complained of sleep disturbances. Nearly 31.67% patients had a positive family history. The pale nasal mucosa was seen in 63.33% patients. Almost 70% patients had intermittent AR, of which 43.33% had a mild degree. About 55% cases had AEC <350 cells/cumm. Almost 51.67% patients had more than five eosinophils per high power field in the biopsy of their inferior turbinates. Conclusion:Although most patients presented with nasal symptoms, ophthalmic and pharyngeal symptoms were commonly presented. ITH with pale nasal mucosa were the most common examination findings. Although AEC was not raised in most of the patients, more than half patients had a raised eosinophil count in the inferior turbinate biopsy. Thus, in a patient with AR, special attention has to be given to the ear, throat, eye, and asthmatic aspect. Eosinophil count in inferior turbinate biopsy is fairly reliable indicator in allergic turbinates.

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“…Adenoid hipertrofisi (AH); kronik havayolu obstrüksiyonuna yol açarak burun tıkanıklığı, kronik ağız solunumu, nazone konuşma, özel yüz görünü-mü, iştahsızlık, okul performansında azalma, horlama, obstrüktif uyku apnesi ve kor pulmonaleye kadar gidebilen klinik tablolara yol açabilir. Medikal tedaviye yanıt vermeyen kronik adenoidit, kronik sinüs enfeksiyonları, yineleyen ya da kronik efüzyonlu otit durumlarında adenoidektomi önerilmektedir (1,2) . Adenoid hipertrofisine yol açan nedenlerin başında kronik adenoid infeksiyonları ve alerjik inflamasyon dolayısıyla alerjik rinit (AR) gelmektedir.…”

Section: Introductionunclassified

Does mold sensitivity cause adenoid hypertrophy in children?

Berksoy¹,

Asilsoy²,

Bağ³

et al. 2017

BUCH

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ÖZAmaç: Adenoid hipertrofisine kronik adenoid infeksiyonları ve alerjik inflamasyon yol açmaktadır. Alerjik inflamasyon dolayısıyla alerjik rinitin önemli nedenlerinden biri başta ev tozu ve küf mantarı olmak üzere ev içi alerjenlerdir. Bu çalışmada, adenoid hipertrofisi gelişen çocuklarda küf mantarı duyarlılığının araştırılması amaçlan-mıştır. Yöntem: Dr. Behçet Uz Çocuk Hastanesi Çocuk Alerji Polikliniğine başvuranlardan öyküsünde adenoidektomi uygulanmış olan 34 hasta çalışmaya alınmıştır. Hastalara önce yakınmalarını ve risk faktörlerini sorgulayan bir anket yapılmış, daha sonra serum total IgE, serum sIgE(mold) düzeyleri ve alerji deri prick test istenmiştir. Bulgular: Çalışmaya alınan hastaların yaşları 5 ile 9 yaş arasında değişmekte olup, 7 (%21)'si kız, 27 (%79)'si erkektir. En sık yakınma; burun tıkanıklığı (%88), horlama (%82) ve terleme (%76), en sık saptanan risk faktörü ise, çevresel sigara dumanı maruziyeti (%52), klima ile ısınma (%38) ve soğuk buhar cihazı kullanımı (%32) olarak bulunmuştur. Hastaların %12'sinde sIgE (Alternaria) pozitifliği, %18'inde alerji deri testinde küf mantarı (Alternaria) duyarlılığı saptanmıştır. Sonuç: Adenoid hipertrofisi olan çocuklarda küf mantarı duyarlılığı araştırılmalı-dır.Anahtar kelimeler: Adenoid, alerji, çocuk, küf mantarı ABSTRACT Objective: Chronic adenoid infections, and allergic inflammation lead to development of adenoid hypertrophy. One of the major causes of allergic rhinitis due to allergic inflammation is indoor allergens, mainly house dust mites and molds. In this study, it was aimed to investigate the frequency of mold fungi sensitivity in the etiology of allergic rhinitis caused by adenoid hypertrophy in children. Methods: Among the patients who had been admitted to Pediatric Allergy Outpatient Clinic of Dr. Behçet uz Children Hospital, 34 cases with adenoidectomy were recruited. First, a questionarre including the risk factors and the symptoms were applied to the patients. Later, total eosinophil counts, serum total IgE and spesific IgE (m6) levels of the patients were examined and finally skin prick test was applied. Results: The age of the patients included in the study varied between five and nine years, seven (21%) of the patients were female and 27 (79%) of them were male. The the most frequent symptoms were nasal congestion (88%), snoring (82%), sweating (76%); the most frequently identified risk factors were enviromental smoke exposure (52%), use of air conditioning for warming (38%), and cold steam device (32%) respectively. Specific IgE (Alternaria) positivity was detected in 12% of the patients and mold sensitivity (Alternaria) in allergy skin test was detected in 18% of the patients. Conclusion: In children with adenoid hypertrophy,mold sensitivity should be investigated.

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The risk of adenoid hypertrophy in children with allergic rhinitis

Cited by 96 publications

References 16 publications

Allergic Rhinitis and its Impact on Asthma (ARIA) 2008*

Allergic Rhinitis and its Impact on Asthma (ARIA) 2008*

A Clinicopathological Study of Allergic Rhinitis

Does mold sensitivity cause adenoid hypertrophy in children?

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