The Relationship Between Biochemical Variables and the Quality of Life in Patients with Chronic Heart Failure

Pleşoianu, Carmen Elena; Andriescu, Gabriela; Șalaru, Delia Lidia; Georgescu, Cătălina Arsenescu

doi:10.37358/rc.17.10.5904

Cited by 6 publications

(8 citation statements)

References 12 publications

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“…Subsequently it was observed that serum CgA increases in patients with pheos/EAPs, as well as in other hormone secreting or non-hormone secreting neuroendocrine tumors such as gastroenteropancreatic tumors, medullary thyroid carcinoma, pituitary tumors (except of prolactinomas), neuroblastomas (Plesoianu et al 2017). The largest amounts of CgA are within the neuroendocrine cells of the adrenal medulla and in the storage granules of the sympathetic nerves (Mirica et al 2018).…”

Section: Biochemical Testsmentioning

confidence: 99%

“…The largest amounts of CgA are within the neuroendocrine cells of the adrenal medulla and in the storage granules of the sympathetic nerves (Mirica et al 2018). Proton pump inhibitors can raise the levels of CgA to 2-3 times (Gut et al 2016) other conditions that can raise the CgA levels are hepatic and cardiac insufficiency, kidney dysfunction, rheumatoid arthritis, inflammatory bowel disease, and atrophic gastritis (Plesoianu et al 2017).…”

Section: Biochemical Testsmentioning

confidence: 99%

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Pheochromocytoma

Farrugia¹,

Anestis

2019

Endocrine Regulations

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Pheochromocytomas are rare tumors originating in the adrenal medulla. They may be sporadic or in the context of a hereditary syndrome. A considerable number of pheochromocytomas carry germline or somatic gene mutations, which are inherited in the autosomal dominant way. All patients should undergo genetic testing. Symptoms are due to catecholamines over production or to a mass effect. Diagnosis is confirmed by raised plasma or urine metanephrines or normetanephrines. Radiology assists in the tumor location and any local invasion or metastasis. All the patients should have preoperative preparation with α-blockers and/or other medications to control hypertension, arrhythmia, and volume expansion. Surgery is the definitive treatment. Follow up should be life-long.

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Section: Biochemical Testsmentioning

confidence: 99%

Section: Biochemical Testsmentioning

confidence: 99%

Pheochromocytoma

Farrugia¹,

Anestis

2019

Endocrine Regulations

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“…Nearly half (49.6%) of the patients with chronic heart failure included in our study, hospitalized for cardiac failure decompensation had at least moderate renal impairment, evaluated by GFR<60mL/min/1.73m 2 . It is thus confirmed that renal dysfunction has a high prevalence in patients with severe heart failure, data from the literature suggesting an estimated prevalence of renal dysfunction in patients with heart failure between 30% and 54% and increase cardiovascular risk and reduces the quality of life [14][15][16][17][18][19][20][21]. Conversely, cardiac disease is a very common comorbidity not only in patients with COPD [22] but also in cases with chronic kidney disease (CKD).…”

Section: Resultsmentioning

confidence: 93%

Renal Dysfunction - a Possible Marker of Severity of Heart Failure

Zaharie¹,

Cârstea²,

Streba³

et al. 2018

Rev. Chim.

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Heart failure (HF) and renal dysfunction are frequent associated in the same patient. The purpose of our study was to assess the prevalence of renal dysfunction and the clinical status in admitted patients for decompensated HF. Material and Methods. 397 patients succesively hospitalized for decompensated HF, NYHA III or IV functional class, with left ventricular ejection fraction (LVEF) � 45% were included in the study. Renal dysfunction was defined by glomerular filtration rate (GFR) [ 60 mL/min/1.73 m 2. The mean GFR in patients with HF was 63.89 � 21.5 mL/min/1.73 m2 .The prevalence of renal dysfunction was 49.6%. Patients with GFR [ 60 mL/min/1.73m2, compared with those with preserved renal function were significantly more frequent older (75.37 � 6.84 vs. 71.33 � 8.08 years; p [0.001), females (53,8% vs. 43.5%; p = 0.04), had a significantly higher prevalence of diabetes mellitus (50.2% vs. 28.5%; p [0.001), atrial fibrillation (53.8% vs 46.2%, p = 0.04) and anemia (47.7% vs. 29.5% ; p [0.001). Also, patients with renal dysfunction had more severe HF than those without renal dysfunction (NYHA class IV: 65% vs 45%, p [0.001, clinical congestion: 78.2% vs 68%, p = 0.02, LVEF [35%: 47.21% vs � 35%, p [0.001). Renal dysfunction can be considered an additional marker of severe cardiac dysfunction along with NYHA IV class and low LVEF. The presence of both renal dysfunction and anemia could represent prognostic markers in HF patients with reduced LVEF.

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“…The physico-chemical theory of inflammation also has an important shortcoming in that it attempts to reduce the inflammatory process to the laws of physical chemistry and colloidal chemistry [34][35][36]. Such an approach to the problem ignores the biological essence of the inflammatory reaction, and therefore has a mechanistic character [37]. From the point of view of physicochemical theory, inflammation remains an exclusively local process, overcoming the role of regulator y systems and mechanisms in the occurrence of the inflammation process, and especially the importance of the nervous system.…”

Section: Developing Physical Chemistry and Biological Chemistry In Thmentioning

confidence: 99%

Inflamatory Chemistry and Organic Surgery in the Maxilar Compression

Craiu¹,

Budacu²,

Dănilă³

et al. 2019

Rev. Chim.

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The morphology and physiology of odonto-periodontal units, muscles, temporomandibular joints, maxillary bones, ligaments are in a constant balance, even if all these components are subject to continuous transformation. Affecting a systemic element will attract imbalances to other levels, and the therapeutic solution will require knowledge and understanding of the ensemble. A complete dental exam must allow identification of all factors responsible or likely to cause imbalance in the stomatognat system. The maxillary compression is an abnormality characterized by a transversal developmental deficiency in the upper jaw and / or the mandibular, showing morpho-functional changes that denote a complex disruption in the development of the dento-maxillary apparatus. The common modification of all the fractured cases is the insufficient development in a transversal sense, dental arcades (mono - or bimaxilar), along with facial and maxillary bone changes, disturbances of inter - maxillary or occlusion blocking, functional deficits and disturbances. The strictly mandibular compression situation is very rare and addresses cases by affecting growth at the condylian level. The study includes 27 cases with jaw compression and benefited from orthognat surgery in the Maxillofacial Surgery Clinic between 2015 - 2018. If untreated, the abnormality determines the appearance of many cavities especially in the front teeth, difficulty in performing dental hygiene will lead to chronic marginal periodontitis, the occurrence of abnormal contact between teeth with joint pain, decreased chewing field. The jaw compression is revealed by multiple facial and oral signs, as well as by functional changes. Orthognatic surgery is the only effective treatment for many patients in jaw compression.

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The Relationship Between Biochemical Variables and the Quality of Life in Patients with Chronic Heart Failure

Cited by 6 publications

References 12 publications

Pheochromocytoma

Pheochromocytoma

Renal Dysfunction - a Possible Marker of Severity of Heart Failure

Inflamatory Chemistry and Organic Surgery in the Maxilar Compression

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