The Rehabilitation of Anton Syndrome

Eby, Sarah A.; Buchner, Eric J.; Bryant, Mary G.; Mak, Henry K.F.

doi:10.1016/j.pmrj.2011.12.012

Cited by 13 publications

(12 citation statements)

References 6 publications

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“…Anton–Babinski syndrome is one of specific form of cortical blindness. The aetiologies include the following: ischemia stroke or haemorrhage involving occipital lobes; posterior leukoencephalopathy induced by chemotherapy, radiotherapy, preeclampsia, adrenoleukodystrophy, or mitochondrial encephalomyopathy, lactic acidosis, and stroke‐like episodes; obstetric haemorrhage with posterior circulation hypoperfusion; and trauma‐related optic neuropathy, bifrontal contusions, and callosal disconnection . Recovery of visual function can be expected if the underlying factor is posterior reversible leukoencephalopathy or cortical hypoperfusionis, and it is corrected quickly .…”

Section: Discussionmentioning

confidence: 99%

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Anton–Babinski syndrome in an old patient: a case report and literature review

et al. 2014

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Anton-Babinski syndrome is a rare disease featuring bilateral cortical blindness and anosognosia with visual confabulation, but without dementia or any memory impairment. It has a unique neuropsychiatric presentation and should be highly suspected in those with odd visual loss and imaging evidence of occipital lobe injury. In the case discussed herein, a 90-year-old man presented with bilateral blindness, obvious anosognosia, and vivid visual confabulation, which he had had for 3 days. Brain computed tomography demonstrated recent hypodense infarctions at the bilateral occipital lobes. Thus, the patient was diagnosed with Anton-Babinski syndrome. Because of his age and the thrombolytic therapy during the golden 3 hours after ischemic stroke, the patient received aspirin therapy rather than tissue plasminogen activator or warfarin. He gradually realized he was blind during the following week, but died of pneumonia 1 month later. In the literature, it is difficult to establish awareness of blindness in patients with Anton-Babinski syndrome, but optimistically, in one report, a patient was aware of blindness within 2 weeks, without vision improvement. Our case illustrates that elderly patients with Anton-Babinski syndrome can partially recover and that 1 week is the shortest time for the establishment of awareness of blindness for sufferers without vision improvement.

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Section: Discussionmentioning

confidence: 99%

“…10,11,14 The first task in rehabilitating such patients is establishing awareness of blindness. 11,13 However, in the literature, it is difficult to establish awareness of blindness in patients with Anton-Babinski syndrome, but optimistically, in one report, a patient was aware of blindness within 2 weeks, without vision improvement (no. 4).…”

Section: Discussionmentioning

confidence: 99%

Anton–Babinski syndrome in an old patient: a case report and literature review

et al. 2014

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“…Once the patient was diagnosed with Anton's syndrome, the rehabilitation strategies must be retaught meticulously. Rehabilitation of cortical blindness alone is a challenge in itself, but with anosognosia, the challenge intensifies as the patients often do not believe that they have a deficit that requires attention [4]. The patient was subjected to a mixture of compensatory and restorative strategies delivered through the combined efforts of rehabilitation medicine physicians, physiotherapists, and occupational therapists.…”

Section: Discussionmentioning

confidence: 99%

Post-traumatic Anton’s Syndrome and Its Rehabilitation Challenges: A Case Report

Andiappan

2021

IRJ

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Objectives: Anton’s syndrome is a rare clinical phenomenon characterized by acquired cortical blindness without the self-awareness of blindness. Patients with this syndrome tend to deny their visual deficits. The literature on the rehabilitation of individuals with Anton’s syndrome is sparse. Methods: We describe a 26-year-old patient who developed Anton’s syndrome in the background of anterior visual tract damage and multiple intracranial bleedings after traumatic brain injury. He presented with visual agnosia and confabulations that were only detected 2 months after trauma. He received 4 months of multidisciplinary rehabilitation care as an outpatient. His comprehensive program consisted of (a) compensatory strategies, (b) restorative strategies, and (c) counseling sessions. Although he showed significant improvement in terms of functional gain with rehabilitation, he still had visual deficits with occasional visual anosognosia that required 24-h supervision. Conclusion: Anton’s syndrome in traumatic brain injury should not be overlooked. A prompt diagnosis is vital to initiate appropriate rehabilitation measures.

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“…We would like to commend Eby et al [1] regarding their recent case description in “The Rehabilitation of Anton Syndrome” and would like to share our perspective related to a review of Anton's original description. Many authors have used the eponym “Anton syndrome” to describe cortical blindness with visual anosognosia.…”

mentioning

confidence: 99%

“…Many authors have used the eponym “Anton syndrome” to describe cortical blindness with visual anosognosia. Specifically, Eby et al [1] state that “Anton syndrome, also referred to as visual anosognosia, is a rare stroke syndrome that involves the bilateral occipital lobes seen most commonly as a result of cerebrovascular disease.” This description is consistent with many recent descriptions of this syndrome.…”

mentioning

confidence: 99%