“…Even assuming fixed intramyocardial conduction velocity, the larger the size of the LV, the longer it would take to activate it. Additionally, adverse remodeling results in impaired intraventricular conduction withing the working myocardium prolonging QRS duration, a mechanism applicable on top of presence of LBBB but also seen as non-specific intraventricular conduction delay when left bundle branch conduction is intact [ 34 , 35 ]. On the other hand, LV dilation may not have a consistent effect on QRS voltage.…”