The Possible Associations of Septal Deviation on Mastoid Pneumatization and Chronic Otitis

Abstract

Our findings suggest that mastoid cell volumes tend to be larger at the contralateral side of the severe septum deviations.

Temporal bone thickness and texture are major determinants of the high rate of insonation failures of transcranial doppler in amerindians (the Atahualpa Project)

“…3 In a poststudy analysis, we found that 9 of 16 (56%) bones showing air in the temporal squama had suboptimal/ absent windows irrespective of bone thickness or sex. As this phenomenon might be associated with infections of the middle ear and with nasal deflection, 14 we interviewed these individuals for history suggestive of chronic otitis and looked for septal deviations on CT scans. Four of 11 cases had antecedents of suppurative otitis and 2 of them had severe nasal deflections with the convex side of the septal curvature contralateral to the abnormally pneumatized temporal bone (Figure 3).…”

Effect of Nasal Septal Deviation on Pneumatization of the Mastoid Air Cell System: 3D Morphometric Analysis of Computed Tomographic Images in a Pediatric Population

“…This finding suggests that distorted air flow owing to NSD can hinder the aeration of the maxillary sinus and mastoid air cell, which is partly consistent with previous reports. [9,13,14] However, this study showed that the degree of NSD (NSA and NSD/ NC ratio) did not correlate with the volumes of the maxillary sinus and mastoid air cell. This finding suggests that the aeration of the maxillary sinus and mastoid air cell may be not simple and has several factors, in addition to NSD.…”

“…They revealed significant differences between deviated and contralateral maxillary sinus volume and concluded that maxillary sinus volume tend to be higher at the contralateral side of severe NSD in adults. Kapusuz Gencer et al [14] evaluated the role of NSD on the volume of mastoid air cells and a possible relationship to chronic otitis, which was conducted in an adult population. They concluded that mastoid cell volumes tend to be larger at the contralateral side of severe NSD in adults.…”

Assessment of nasal airway resistances and nasal mucociliary activities of patients with unilateral chronic otitis media by acoustic rhinometry and saccharin test

“…When this mucosa is subjected to edema or negative intratympanic pressure, spreading of infectious events in the nasopharynx into the middle ear becomes easier. In consequence, otitis media develops [1,4]. Also, reflux of nasopharyngeal content into the middle Correspondence ear through the eustachian tube can produce direct mechanical damage to the middle ear mucosa and, eventually, middle ear inflammation.…”