Patients with functional gait disorder (FGD) are often referred for physiotherapy, but data on their outcome is limited. The authors present a case series of 35 patients who received targeted physiotherapy for FGD at a regional neuroscience center (mean number of sessions, 11). Significant improvements in the Modified Rivermead Mobility Index (score range, 0-40) were recorded between pretreatment and posttreatment (mean pretreatment vs. posttreatment score, 20 vs. 37, respectively). Improvements also were seen in patients who had chronic symptoms, including those with symptom duration over 6 months (mean pretreatment vs. posttreatment score, 21 vs. 33, respectively) and in patients who had no psychological intervention. These data support the hypothesis that specific physiotherapy for FGD can be surprisingly effective and further encourage the development of larger randomized trials to test efficacy.There is increasing interest and data are accumulating on the outcomes of patients with functional (psychogenic) movement disorder (FMD) who receive physiotherapy. In collaboration with Glenn Nielsen and colleagues in London, we previously described in detail the content of FMD-specific physiotherapy 1 This emphasizes the importance of understanding the reversible nature of the diagnosis and sharing evidence for this, such as Hoover's sign and the entrainment test, with the patient. Physiotherapy for many neurologic diseases, such as stroke, relies on focused attention on the poorly functioning limb. For patients with FMD, attention away from the limb may be more appropriate. The aim is to "train" patients away from habitual abnormal movements back to more normal automatic movements.We describe our experience prospectively evaluating a series of 35 patients treated by 2 of us (A.M. and M.B.) in a regional neuroscience center using these principles to add to the literature in this area.
Case SeriesWe carried out a prospective study of patients seen between November 2012 and December 2014 with disabling FGD (predominantly associated with limb weakness) who were referred to A.M. or M.B. for inpatient physiotherapy in an acute neurology ward in the regional neuroscience service in Glasgow. All patients had been diagnosed with FGD by a consultant neurologist, most with extensive supplementary radiologic and neurophysiological testing. Some were admitted acutely because of their symptoms. Most were subacute or chronic presentations who had been admitted for investigation and treatment by their treating neurologist. There was no formal triage process. We recorded demographic data, duration of motor symptoms, and Modified Rivermead Mobility Index (MRMI) scores (before treatment and immediately after the end of treatment). Approval by an ethics committee was not applicable, because the study was carried out as part of routine care.