“…Analyses of DNA sequences located in the vicinity of chromosomal translocation breakpoints, in some kinds of tumors, have suggested that homologous recombination and/or site-specific recombinogenic sequences may be involved. These include, for example, heptamer/ nonamer sequences, Translin consensus sequences, topoisomerase I and II clevage sequences, chi-consensus sequences, Alu repeats, palindromic sequences, alternating purine/pyrimidine and polypurine or polypyrimidine sequences (Bullock et al, 1985;Finger et al, 1986;Krowezynska et al, 1986;Nicholls et al, 1987;Boehm et al, 1989;Chen et al, 1989;Sperry et al, 1989;Camerini-Otero and Hsieh, 1993;Aoki et al, 1995;Broeker et al, 1996).…”