The microbiology of the acute dental abscess

Abstract

The acute dental abscess is frequently underestimated in terms of its morbidity and mortality. The risk of potential serious consequences arising from the spread of a dental abscess is still relevant today with many hospital admissions for dental sepsis. The acute dental abscess is usually polymicrobial comprising facultative anaerobes, such as viridans group streptococci and the Streptococcus anginosus group, with predominantly strict anaerobes, such as anaerobic cocci, Prevotella and Fusobacterium species. The use of non-culture techniques has expanded our insight into the microbial diversity of the causative agents, identifying such organisms as Treponema species and anaerobic Gram-positive rods such as Bulleidia extructa, Cryptobacterium curtum and Mogibacterium timidum. Despite some reports of increasing antimicrobial resistance in isolates from acute dental infection, the vast majority of localized dental abscesses respond to surgical treatment, with antimicrobials limited to spreading and severe infections. The microbiology and treatment of the acute localized abscess and severe spreading odontogenic infections are reviewed.

The Lung Microbiome in Moderate and Severe Chronic Obstructive Pulmonary Disease

“…Coribacteriales, a member of the gram-positive phylum Actinobacteria, were increased in the COPD and LLQ analyses. This increase was extended down to the genus level: Atopobium, a vaginal commensal and member of the oral flora [21] and Cryptobacterium, a cause of dental abscess [22]. Two orders in the grampositive phylum Firmicutes were differentially abundant.…”

Cariogenic microflora and pH in superficial and deep layers of occlusal carious lesions - A metagenomic analysis

“…The dominant phyla in the dentine caries lesions usually include Firmicutes with Lactobacillus accounting for nearly 40% of the total general. [28] The occurrence of Lactobacillus in saliva is common in high caries patient. Lactobacillus metabolizes dietary sugars immediately and produces acid which is cause decalcification of teeth.…”

Periapical Abscess – A Review

“…Recent advances in microbiological testing have resulted in the discovery of other pathogens as causative agents of this type of infection, including Treponema spp., Atopobium, Bulleidia extructa and Mogibacterium species, as well as Cryptobacterium curtum. [4] Up to a third of the microorganisms isolated in these cases produce beta-lactamases, which significantly reduces treatment options.…”