The influence of age on the fusion frequency of flicker.

Simonson, Ernst; Enzer, Norbert; Blankstein, Samuel S.

doi:10.1037/h0056872

Cited by 33 publications

(13 citation statements)

References 3 publications

(2 reference statements)

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“…Apart from partial disagreements with respect to specific age ranges (1,6,16) the general pattern of our growth and decline curves is in good agreement with most previous studies of importance (5,9,11,13). Hence we felt justified in using our results to establish baseline norms for evaluating the same processes in psychotic patients.…”

Section: Discussionsupporting

confidence: 83%

“…Sex differences in means were not significant except in the age range 60 to 80 years (1 per cent level). Whereas investigators (2,10,11,16) generally agree that the correlation between critical flicker-fusion frequency and age tends to be negative, there is some disagreement (6,12) as to whether a very early increase in this function takes place. As Figure 4 indicates, our sample showed a definite rise in CFF values between the childhood (a) and early adolescent (b) groups (significant at the 1 per cent level).…”

Section: Resultsmentioning

confidence: 99%

See 1 more Smart Citation

Developmental norms on four psychophysiological measures for use in the evaluation of psychotic disorders.

Jz¹,

He²

1958

Canadian Journal of Psychology / Revue canadienne de psychologi

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Section: Discussionsupporting

confidence: 83%

Section: Resultsmentioning

confidence: 99%

Developmental norms on four psychophysiological measures for use in the evaluation of psychotic disorders.

Jz¹,

He²

1958

Canadian Journal of Psychology / Revue canadienne de psychologi

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“…In 1941 Simonson, Enzer and Blankstein (25) used 47 Ss varying in age from 10 to 80. They divided the Ss into four groups: group one (10-19 years of age) contained four Ss, group two (20-29) 18, group three (30-39) 10, and group four (over 40) 15.…”

Section: A Introductionmentioning

confidence: 99%

Age and sex differences in critical flicker frequency.

Misiak

1947

Journal of Experimental Psychology

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“…ment is promoted by the male sex hormones in animals (31) and in man (32)(33)(34). Various tests, objective in character, have shown a measurable increase in muscular performance in human subjects, following the administration of male hormone.…”

Section: Effects Of the Administration Of Testosterone Propionate--mmentioning

confidence: 99%

The Synthesis, Storage, and Excretion of Creatine, Creatinine, and Glycocyamine in Progressive Muscular Dystrophy and the Effects of Certain Hormones on These Processes

Hoagland

Gilder

Shank

1945

Journal of Experimental Medicine

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The diminished excretion of creatinine in progressive muscular dystrophy is a more striking and specific phenomenon than the excess excretion of creatine, marked though this is. While creatinuria is invariably encountered in all cases of long-standing dystrophy, the extent to which the excretion of creatinine is decreased provides a more reliable indication of the severity of the disease since an excess output of creatine may occur physiologically in normal human subjects and in many pathological conditions not known to be associated with muscle disease. In progressive muscular dystrophy the residual muscle mass, as inferred from the excretion of creatinine, provides a useful index of the state of the disease at any given time. Although there is excessive creatinuria in progressive muscular dystrophy, there is no evidence that a deprivation of methyl stores occurs through a loss of urinary creatine. The loss of methyl groups contained in the excess creatine is, under ordinary conditions of diet, almost exactly compensated for by a drop in the excretion of methyl groups in the urinary creatinine. Testosterone propionate, administered over variable periods of time, resulted in the retention of creatine both in normal male children and in male children with progressive muscular dystrophy, as shown in the normal subjects by a diminution in creatine output, and in both by an excess creatinuria for variable periods of time following withdrawal of the hormone. An increase in the excretion of creatine in progressive muscular dystrophy occurred following the administration of methyl testosterone. Neither testosterone propionate nor methyl testosterone appeared to effect any consistent change in the output or urinary creatinine. No effects on the excretion of creatine and creatinine were observed following the prolonged administration of concentrate of gonadotropic and thyrotropic principles of the hypophysis, or from the administration of desoxycorticosterone acetate to patients with progressive muscular dystrophy. Except in one case, in which marked improvement was observed following the administration of testosterone propionate, no effects on the clinical course of the patients with progressive muscular dystrophy were observed as a result of treatment by any of the various hormones employed in this study.

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The influence of age on the fusion frequency of flicker.

Cited by 33 publications

References 3 publications

Developmental norms on four psychophysiological measures for use in the evaluation of psychotic disorders.

Developmental norms on four psychophysiological measures for use in the evaluation of psychotic disorders.

Age and sex differences in critical flicker frequency.

The Synthesis, Storage, and Excretion of Creatine, Creatinine, and Glycocyamine in Progressive Muscular Dystrophy and the Effects of Certain Hormones on These Processes

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