The incidence and mortality of intraventricular conduction defects in acute myocardial infarction

Col, Jacques; Weinberg, Sylvan Lee

doi:10.1016/0002-9149(72)90529-2

Cited by 147 publications

(42 citation statements)

References 20 publications

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“…Left anterior hemiblock is the most common conduction disturbance complicating all types of acute infarction,14 19,20 with an incidence ranging from 4% in the series of Scheinman and Brenman" to 15.2% in the series of Marriot and Hogan. '9 Previous studies have not examined the incidence and effects of acute LAH complicating the course of inferior wall infarction alone.…”

Section: Discussionmentioning

confidence: 99%

Incidence and significance of left anterior hemiblock complicating acute inferior wall myocardial infarction.

Kourtesis¹,

Lichstein²,

Chadda³

et al. 1976

Circulation

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SUMMARY The hospital course and serial vectorcardiograms of 56 consecutive patients with acute inferior wall myocardial infarction were reviewed. Left anterior hemiblock (LAH) complicating inferior wall myocardial infarction was diagnosed by vectorcardiographic criteria. Seven patients (12.5%) developed LAH between the first and third hospital day, while 49 patients did not. There was no significant LEFT ANTERIOR HEMIBLOCK (LAH) may be difficult to diagnose in the presence of inferior wall myocardial infarction due to the frequent leftward shift of the frontal plane QRS forces by the infarcted area.' Vectorcardiographic criteria have been described by Castellanos,2 Kulbertus,3 and Benchimol4 which enable the diagnosis of LAH to be made in the presence of inferior wall myocardial infarction. It is the purpose of this study, using these criteria, to examine prospectively patients with inferior wall myocardial infarction and to determine the incidence and clinical significance of the complication of acute LAH. MethodsData were reviewed on 56 consecutive patients admitted to a coronary care unit with acute inferior wall myocardial infarction. Patients with LAH at the time of admission were not included. The criteria for diagnosis were: 1) History of ischemic chest pain.2) Significant Q waves and ST-segment elevation in leads II, III, aVF with reciprocal ST-segment depression in leads I and aVL. a) Associated lateral wall involvement was diagnosed by appearance of a 0.04 sec Q wave and ST elevation in leads V5 and V6. 3) Serial changes of serum glutamic oxaloacetic transaminase, lactic dehydrogenase, and creatinine phosphokinase. All patients were admitted to the coronary care unit within the first 24 hours following onset of acute chest pain and were constantly monitored using a nonfade television and display, and a V, monitor lead. A 12-lead electrocardiogram was recorded daily or more often if indicated clinically. Routine coronary care included a constant intravenous infusion of lidocaine (average 2 mg/min) and heparin 75 mg intravenously every six hours, unless specific difference between these two groups when compared for age, sex, incidence of congestive heart failure, atrial and ventricular arrhythmias, atrioventricular (A-V) block, hospital mortality, and previous hypertension, diabetes mellitus, and myocardial infarction. We conclude that LAH is a relatively common complication of acute inferior wall myocardial infarction, with no apparent effect on the clinical course.contraindications were present in a patient. Vectorcardiograms using the Frank lead system were recorded on day one and day three and on the basis of these studies the patients were separated into two groups.Group I: Patients with conventional frontal plane vectorcardiographic criteria for inferior wall infarction.'-7 a) Superior orientation and clockwise rotation of the initial 25 msec vector. Group II: Patients with frontal plane vectorcardiographic criteria for inferior wall infarction coexisting with LAH.' a) Clockwise and superior inscri...

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Section: Discussionmentioning

confidence: 99%

Incidence and significance of left anterior hemiblock complicating acute inferior wall myocardial infarction.

Kourtesis¹,

Lichstein²,

Chadda³

et al. 1976

Circulation

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show abstract

“…El BAV de alto grado relacionado con infarto anterior se localiza más a menudo bajo el nodo AV w181 . El BAV que complica el infarto agudo de miocardio, a menudo se soluciona por sí solo pasados días o semanas [133][134][135][136] , y sólo el 9% de estos pacientes requieren estimulación cardiaca permanente 132 . Los pacientes con BAV tienen una mortalidad hospitalaria y a los 30 días mayor que aquellos con conducción AV conservada independientemente del sitio del infarto 133 .…”

Section: Estimulación Cardiaca En El Infarto Agudo De Miocardiounclassified

2013 ESC Guidelines on Cardiac Pacing and Cardiac Resynchronization Therapy

Brignole

Auricchio

Barón-Esquivias

et al. 2014

Revista Española de Cardiología (English Edition)

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“…Zaawansowany blok AV związany z zawałem ściany przedniej coraz częściej jest umiejscowiony poniżej . Blok AV w przebiegu ostrego zawału serca najczęściej ustępuje samoistnie w ciągu kilku dni lub tygodni [133][134][135][136] i tylko 9% chorych wymaga stałej stymulacji [132]. Pacjenci z blokiem AV mają wyższą śmiertelność szpitalną i 30-dniową niż chorzy z zachowanym przewodzeniem AV, niezależnie od lokalizacji zawału [133].…”

Section: Stymulacja W Ostrym Zawale Sercaunclassified

Wytyczne ESC dotyczące stymulacji serca i terapii resynchronizującej w 2013 roku

Brignole

Auricchio²,

Barón-Esquivias³

et al. 2013

Kardiol Pol

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The incidence and mortality of intraventricular conduction defects in acute myocardial infarction

Cited by 147 publications

References 20 publications

Incidence and significance of left anterior hemiblock complicating acute inferior wall myocardial infarction.

Incidence and significance of left anterior hemiblock complicating acute inferior wall myocardial infarction.

2013 ESC Guidelines on Cardiac Pacing and Cardiac Resynchronization Therapy

Wytyczne ESC dotyczące stymulacji serca i terapii resynchronizującej w 2013 roku

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