1974
DOI: 10.1259/0007-1285-47-564-875
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The cortical rim sign of renal infarction

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1982
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Cited by 21 publications
(4 citation statements)
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“…2 The presence of penetrating vessels from the capsular plexus spares the cortex from infarction, resulting in the classical cortical rim sign. 5 Anticoagulant therapy may lead to resolution of the infarct, and the mass effect may disappear. 6 Chronic hyalinized fibrosis with dystrophic calcification is responsible for the high attenuation values on CT scan.…”
Section: Discussionmentioning
confidence: 99%
“…2 The presence of penetrating vessels from the capsular plexus spares the cortex from infarction, resulting in the classical cortical rim sign. 5 Anticoagulant therapy may lead to resolution of the infarct, and the mass effect may disappear. 6 Chronic hyalinized fibrosis with dystrophic calcification is responsible for the high attenuation values on CT scan.…”
Section: Discussionmentioning
confidence: 99%
“…This is due to the differences in arterial supply to this region of cortex compared with the remainder of the renal parenchyma. 55 Segmental infarcts appear as discrete wedge-shaped areas of nonenhancement compared with the remainder of the normally perfused renal parenchyma. Arterial-phase CT images can demonstrate occluded main or segmental renal arteries but are often not necessary for diagnosis compared with routine portal venous-phase images.…”
Section: Radiation-associated Complicationsmentioning
confidence: 99%
“…Arterial-phase CT images can demonstrate occluded main or segmental renal arteries but are often not necessary for diagnosis compared with routine portal venous-phase images. 19,40,55,56 Ureteral Injuries…”
Section: Radiation-associated Complicationsmentioning
confidence: 99%
“…Segmental absence may be due to renal space-occupying processes, hemodynamic changes in peripheral branches of the renal artery, or small vessel disease such as vasculitides [ 7 , 8 ]. In the setting of renovascular compromise, the rim pattern represents preserved subcapsular perfusion by collateral flow [ 9 , 10 ]. Temporal delay in the progression of the nephrogram may be attributed to a unilateral decrease in renal blood flow [ 11 ].…”
Section: Introductionmentioning
confidence: 99%