1995
DOI: 10.1016/s0733-8651(18)30053-5
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The Cardiovascular Effects of Environmental Traumas

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Cited by 28 publications
(1 citation statement)
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“…Simultaneously, during the early period of burn disease, a decrease in cardiac output occurs, that cannot be influenced by sufficient volume supply [9]. This is possibly induced by circulating humoral factors like tumor necrosis factor (TNF), endothelin-1, interleukins or free oxygen radicals with a significant negative inotropic effect, as well as by a decreased response to endogenous catecholamines and a diminished coronary blood flow [4,6,11,13,14]. Paralleling the decrease in cardiac output, peripheral vascular resistance increases.…”
Section: Posing the Problemmentioning
confidence: 99%
“…Simultaneously, during the early period of burn disease, a decrease in cardiac output occurs, that cannot be influenced by sufficient volume supply [9]. This is possibly induced by circulating humoral factors like tumor necrosis factor (TNF), endothelin-1, interleukins or free oxygen radicals with a significant negative inotropic effect, as well as by a decreased response to endogenous catecholamines and a diminished coronary blood flow [4,6,11,13,14]. Paralleling the decrease in cardiac output, peripheral vascular resistance increases.…”
Section: Posing the Problemmentioning
confidence: 99%