2018
DOI: 10.1016/j.iccn.2017.05.003
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The cardio-respiratory effects of intra-abdominal hypertension: Considerations for critical care nursing practice

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Cited by 10 publications
(13 citation statements)
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“…Some cardiac signs of raised intra-abdominal pressure are abnormal increase in pulmonary artery pressure, central venous pressure increase, cardiac output decrease, increased heart rate, variable MAP, pulmonary artery wedge pressure increase, systemic vascular resistance increase, left ventricular end-diastolic pressure decrease and right ventricular end-diastolic pressure decrease. [6] Respiratory changes are functional residual capacity decrease, volume decrease, peak airway pressure increase, mean airway pressure increase, plateau pressures increase, intrinsic PEEP increase, dead-space ventilation increase, pulmonary oedema increase and work of breathing increase. [6] With the acute decompensation and difficulty in ventilating the patient, an aggressive decompression laparotomy was deemed to be the most effective treatment option.…”
Section: Discussionmentioning
confidence: 99%
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“…Some cardiac signs of raised intra-abdominal pressure are abnormal increase in pulmonary artery pressure, central venous pressure increase, cardiac output decrease, increased heart rate, variable MAP, pulmonary artery wedge pressure increase, systemic vascular resistance increase, left ventricular end-diastolic pressure decrease and right ventricular end-diastolic pressure decrease. [6] Respiratory changes are functional residual capacity decrease, volume decrease, peak airway pressure increase, mean airway pressure increase, plateau pressures increase, intrinsic PEEP increase, dead-space ventilation increase, pulmonary oedema increase and work of breathing increase. [6] With the acute decompensation and difficulty in ventilating the patient, an aggressive decompression laparotomy was deemed to be the most effective treatment option.…”
Section: Discussionmentioning
confidence: 99%
“…[6] Respiratory changes are functional residual capacity decrease, volume decrease, peak airway pressure increase, mean airway pressure increase, plateau pressures increase, intrinsic PEEP increase, dead-space ventilation increase, pulmonary oedema increase and work of breathing increase. [6] With the acute decompensation and difficulty in ventilating the patient, an aggressive decompression laparotomy was deemed to be the most effective treatment option. [7] Sonar-guided needle aspiration may have been an option, but this would have required some time to find a sonar to borrow, which may have then compromised the patient.…”
Section: Discussionmentioning
confidence: 99%
“…Increasing the IAP level is expected among the intensive care patients according to many factors especially that affect the stability of abdominal cavity either by the pathology of the disease itself or the treatment modalities. However, the abdominal compliance is limited and is considered intra-abdominal hypertension (IAH) as 12: 20 mmHg and abdominal compartment syndrome (ACS) as more than 21mmHg (Christensen, Craft, & Nursing, 2018). In literature, IAP disturbs the organ tissue perfusion, possibly leading to severe ischemic or circulatory changes.…”
Section: Intra-abdominal Pressure (Iap) Of Enteral-fed Patientsmentioning
confidence: 99%
“…[3,8] Part two: It included abdominal perfusion pressure (APP = MAP -IAP. MAP is the mean arterial pressure.)…”
Section: Tool I: Patients' Assessment Sheetmentioning
confidence: 99%
“…[2] When the pressure is sustained or elevated due to pathological causes above 12 mmHg, developed to Intra-abdominal hypertension (IAH). [3] New organ failure or dysfunction with sustained IAP greater than 20 mmHg (with or without abdominal perfusion pressure [APP] < 60 mmHg) is called abdominal compartment syndrome (ACS). [4,5] This syndrome is responsible for the increase in prevalence of death from 90% to 100% if not immediately recognized and treated.…”
Section: Introductionmentioning
confidence: 99%