The Carbon Dioxide Response Curve for Chronic Hypercapnia in Man

Striho, Van Yperselle de; Brasseur, L.; Coninck, De

doi:10.1056/nejm196607212750301

Cited by 65 publications

(13 citation statements)

References 18 publications

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“…The presence of hypoxemia, potassium depletion, and other abnormalities might well lead to an He-Paco2 relationship notably different from that observed here. It is of interest, however, to note that in the one published study designed to assess the influence of chronic pulmonary insufficiency upon acute changes in arterial carbon dioxide tension the gross behavior appeared to be similar to that which we have found in the dog, i.e., pH was seemingly defended more effectively than in normal volunteers (16 (17)(18)(19)(20).…”

Section: Methodssupporting

confidence: 64%

The influence of graded degrees of chronic hypercapnia on the acute carbon dioxide titration curve

Goldstein¹,

Gennari²,

Schwartz³

1971

J. Clin. Invest.

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Section: Methodssupporting

confidence: 64%

The influence of graded degrees of chronic hypercapnia on the acute carbon dioxide titration curve

Goldstein¹,

Gennari²,

Schwartz³

1971

J. Clin. Invest.

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“…Careful observations of patients with chronic hypercapnia as a result of chronic obstructive pulmonary disease allowed estimation of a mean ⌬[HCO ]/ ⌬PaCO 2 seems to flatten. 8,9 More recently, a substantially larger slope was reported, but the small number of blood gas measurements, one for each of 18 patients, calls into question the validity of the conclusion reached. 10 …”

Section: Magnitude and Time Course Of The Secondary Responsesmentioning

confidence: 99%

Secondary Responses to Altered Acid-Base Status

Adrogué¹,

Madias²

2010

Journal of the American Society of Nephrology

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“…A value of 0.30 was reported by Van Ypersele de Strihou et al [4] in nonselected hypercapnic patients.…”

Section: Resultsmentioning

confidence: 72%

Relationship between Chronic PaCO<sub>2</sub> and Total Body Buffering Capacity in Patients with Chronic Obstructive Lung Disease

Rizzato¹,

Ferrara²,

L³

et al. 1980

Respiration

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17 subjects were carefully selected among 150 patients with COLD: all patients with one or more conditions or diseases that could alter the physiological response to chronic hypercapnia were eliminated. During a period of 13 to 28 days, the acid-base response to slow carbon dioxide changes was studied in the selected patients. Total body buffering capacity was expressed as dH⁺a/dPaCO₂ and showed to be an inverse function of chronic PaCO₂ (defined as the value of PaCO₂ in clinical and acid-base steady status for each patient), according to the previous suggestions of Weiss and Dulfano and to the report of Ingram and co-workers during acute carbon dioxide changes in the same patients

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The Carbon Dioxide Response Curve for Chronic Hypercapnia in Man

Cited by 65 publications

References 18 publications

The influence of graded degrees of chronic hypercapnia on the acute carbon dioxide titration curve

The influence of graded degrees of chronic hypercapnia on the acute carbon dioxide titration curve

Secondary Responses to Altered Acid-Base Status

Relationship between Chronic PaCO<sub>2</sub> and Total Body Buffering Capacity in Patients with Chronic Obstructive Lung Disease

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