“…Increased sympathoadrenal activity may lead to incoordinate uterine action and reduced uteroplacental perfusion. 3 The metabolic outcome is hyperglycaemia with a poor insulin response, lipolysis with increased free fatty acids, ketones and lactate. Such acids cross the placenta and together with catecholamines, increase fetal oxygen requirement, so maternal metabolic acidosis from this further cause is compounded in the baby.…”