The Artery Wall and the Pathogenesis of Progressive Atherosclerosis

Wissler, Robert W.

doi:10.1007/978-1-4612-6071-4_83

Cited by 6 publications

(1 citation statement)

References 22 publications

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“…These focal lesions, which resemble the fibromuscular lesions in atherosclerosis might be a result of monoclonal proliferation of the smooth muscle cell. This is one among other theories which could explain the development of the atherosclerotic plaque (Bjorkerud 1979, McGill 1977, Ross 1980, Wissler 1980. Evidence for a monoclonal origin of the human atherosclerotic plaque, fibromuscular lesions and fatty streaks, seem well established by studies on the X-linked glucose -6phosphate dehydrogenase (Benditr and Bendirt , Benditr 1974, Pearson er al.…”

Section: Discussionmentioning

confidence: 90%

Ultrastructure of the Umbilical Artery From a Newborn Delivered at Term by a Mother Who Smoked 80 Cigarettes Per Day

Asmussen

1982

Acta Pathologica Microbiologica Scandinavica Series A :Patholog

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Asmussen, 1. Ultrastructure of the umbilical artery from a newborn delivered at term by a mother who smoked 80 cigarettes per day. Acta path. microbiol. immunol. scand. Sect. A, 90: 397-404, 1982. The ultrastucture was studied of an umbilical artery from a newborn child delivered by a healthy mother who smoked as many as 80 cigarettes per day. Focal areas in the luminal part of the media appeared acellular consisting of collagen fibers only. In the deeper parts of the media the smooth muscle cells were hypertrophic and showed sign of proliferation. These smooth muscle cells had accumulated huge amounts of glycogen in the perinuclear region. The glycogen accumulations were always found in close connection with the mitochondria and often associated with lipid droplets.Occasionally phagocytosis and degradation of collagen by the smooth muscle cells were visualized.

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Section: Discussionmentioning

confidence: 90%