“…These electrophysiological differences are congruent with neuroanatomical features associated with ASD (Nair, Treiber, Shukla, Shih, & Müller, ). One hypothesis is that the differences in the thalamocortical system between ASD and healthy populations indicate atypical connectivity (Ferradal et al, ; Woodward, Giraldo‐Chica, Rogers, & Cascio, ) and functioning of thalamocortical neural substrates in ASD (Daoust, Limoges, Bolduc, Mottron, & Godbout, ; Rochette et al, ), and result in atypical processing of cognitive, attentional, and sensorimotor pathways in children and adults with ASD (Nair et al, ). Thalamocortical projections increase over the beginning years of life (Alcauter et al, ), and alterations in the thalamocortical connections such as under connectivity to prefrontal, parieto‐occipital, and temporal regions (Nair et al, ) suggest atypical connectivity of the developing brain.…”