Temporary trigeminal disorder as a result of pneumocephalus after subarachnoid block

Cosío, Francisco G.; Bermejo-Alvarez, M A; Fervienza, P; Jimenez, Lia; Castañón, Eduardo; Díaz, M.L.

doi:10.1093/bja/aeg185

Cited by 5 publications

(5 citation statements)

References 17 publications

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“…As the sensory block level was not higher than T6, her difficulty in speaking was not due to recurrent laryngeal nerve palsy. Temporary trigeminal disorder may occur as a result of pneumocephalus after subarachnoid block [10]. In the present case, no air bubble was found on MRI scan.…”

Section: Discussioncontrasting

confidence: 38%

Trigeminal nerve and facial nerve palsy after combined spinal-epidural anesthesia for cesarean section

Fang

Lin

Qiang

et al. 2010

Journal of Clinical Anesthesia

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Section: Discussioncontrasting

confidence: 38%

Trigeminal nerve and facial nerve palsy after combined spinal-epidural anesthesia for cesarean section

Fang

Lin

Qiang

et al. 2010

Journal of Clinical Anesthesia

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“…Vagal traction may stimulate chemoreceptors in the medulla to induce nausea. Traction on the abducens and trochlear nerves can lead to ocular muscle paralysis, with subsequent strabismus and double vision [10,11]. Hearing loss has also been reported, due to a low CSF pressure transmitted to the inner ear via the cochlear aqueduct.…”

Section: Pathophysiology Of Plphmentioning

confidence: 99%

Lumbar Puncture: It Is Time to Change the Needle

2010

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Lumbar puncture is a frequent procedure performed by physicians from several disciplines to help establish a diagnosis and treatment for several diseases. Post-lumbar puncture headache (PLPH) is a frequent complication that typically lasts for a couple of days and can be severe enough to immobilize the patient and to require therapy. There are several risk factors identified, pain characteristics, and characteristic findings on spinal and head magnetic resonance imaging. There are several procedural factors that have been identified to be of consequence in attenuating the PLPH incidence, specifically the needle type and size used for this procedure. Once PLPH occurs, the clinician should treat it conservatively with bed rest, analgesics and increased fluids intake, especially caffeine-containing beverages, as it can dramatically affect the patient’s wellness. If the pain is severe and disabling and does not respond to conservative treatment, a blood patch should be considered at least 24–48 h following the LP. Epidural blood patch is a safe and rapidly effective treatment in experienced hands. Furthermore, patients who developed PLPH should be advised to contact the medical staff in case of changes in the characteristics of headaches. When a patient who was diagnosed with PLPH has a change in the pain character, or additional neurological manifestations appear, an urgent brain CT/head MRI should be performed to exclude rarer life-threatening intracranial complications.

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“…This study opens a new option in targeting IL-17A to modulate inflammatory response to CS and may be the bases for new therapy for COPD. chronic obstructive pulmonary disease; interleukin-17 CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) is defined as "a disease state characterized by airflow limitation that is not fully reversible, and that is usually both progressive and associated with an abnormal inflammatory response of the lungs to noxious particles or gases" (13). Caused predominantly by cigarette smoking, COPD is one of the leading causes of mortality globally (37).…”

mentioning

confidence: 99%

Genetic deletion of IL-17A reduces cigarette smoke-induced inflammation and alveolar type II cell apoptosis

Chang

Al-Alwan

Audusseau

et al. 2014

American Journal of Physiology-Lung Cellular and Molecular Phys

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Chronic obstructive pulmonary disease (COPD) is an inflammatory disorder marked by relative resistance to steroids. Inflammation and apoptosis have been suggested to be important mechanisms for COPD. Interleukin (IL)-17 superfamily has been associated with chronic inflammation and diminished responses to steroids. It is reasonable to consider that IL-17 may play a role in the pathogenesis of COPD. In this study, we examined IL-17 expression in mice exposed to cigarette smoke (CS) and investigated the contribution of IL-17 to CS-induced inflammation and alveolar cell apoptosis in IL-17(-/-) mice. After exposing wild-type and IL-17(-/-) mice to mainstream CS for 4 wk, IL-17A, but not IL-17F, expression was increased in mice upon CS exposure. Neutrophil infiltration in the lungs of IL-17(-/-) mice was significantly decreased. In IL-17(-/-) mice, there is reduced expression of IL-6, macrophage inflammatory protein-2, and matrix metalloproteinase-12 compared with wild-type mice after CS exposure. The number of apoptotic type II alveolar cells was significantly increased in CS-exposed wild-type mice but not in IL-17(-/-) mice. The effect of IL-17A on type II alveolar cell apoptosis was confirmed in vitro through either addition of IL-17A or transient knockdown of IL-17A by small-interfering RNA transfection in type II alveolar cells. These findings suggest that IL-17A plays an important role in the inflammatory response to CS exposure through increased multiple inflammatory mediators. Moreover, IL-17 may also contribute to type II alveolar cell apoptosis. This study opens a new option in targeting IL-17A to modulate inflammatory response to CS and may be the bases for new therapy for COPD.

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Temporary trigeminal disorder as a result of pneumocephalus after subarachnoid block

Cited by 5 publications

References 17 publications

Trigeminal nerve and facial nerve palsy after combined spinal-epidural anesthesia for cesarean section

Trigeminal nerve and facial nerve palsy after combined spinal-epidural anesthesia for cesarean section

Lumbar Puncture: It Is Time to Change the Needle

Genetic deletion of IL-17A reduces cigarette smoke-induced inflammation and alveolar type II cell apoptosis

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