Tabernanthalog and ibogainalog inhibit the α7 and α9α10 nicotinic acetylcholine receptors via different mechanisms and with higher potency than the GABAA receptor and CaV2.2 channel

Tae, Han-Shen; Ortells, Marcelo O.; Yousuf, Arsalan; Xu, Sophia Q.; Akk, Gustav; Adams, David J.; Arias, Hugo R.

doi:10.1016/j.bcp.2024.116183

Biochemical Pharmacology

2024

DOI: 10.1016/j.bcp.2024.116183

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Tabernanthalog and ibogainalog inhibit the α7 and α9α10 nicotinic acetylcholine receptors via different mechanisms and with higher potency than the GABAA receptor and CaV2.2 channel

Han-Shen Tae,

Marcelo O. Ortells,

Arsalan Yousuf

et al.

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Cited by 1 publication

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Non-hallucinogenic compounds derived from iboga alkaloids alleviate neuropathic and visceral pain in mice through a mechanism involving 5-HT2A receptor activation

Arias,

Micheli,

Rudin

et al. 2024

Biomedicine & Pharmacotherapy

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Non-hallucinogenic compounds derived from iboga alkaloids alleviate neuropathic and visceral pain in mice through a mechanism involving 5-HT2A receptor activation

Arias,

Micheli,

Rudin

et al. 2024

Biomedicine & Pharmacotherapy

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Tabernanthalog and ibogainalog inhibit the α7 and α9α10 nicotinic acetylcholine receptors via different mechanisms and with higher potency than the GABAA receptor and CaV2.2 channel

Cited by 1 publication

References 27 publications

Non-hallucinogenic compounds derived from iboga alkaloids alleviate neuropathic and visceral pain in mice through a mechanism involving 5-HT2A receptor activation

Non-hallucinogenic compounds derived from iboga alkaloids alleviate neuropathic and visceral pain in mice through a mechanism involving 5-HT2A receptor activation

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